Familial CD8 deficiency due to a mutation in the CD8 alpha gene

J Clin Invest. 2001 Jul;108(1):117-23. doi: 10.1172/JCI10993.

Abstract

CD8 glycoproteins play an important role in both the maturation and function of MHC class I-restricted T lymphocytes. A 25-year-old man, from a consanguineous family, with recurrent bacterial infections and total absence of CD8(+) cells, was studied. Ab deficiencies and ZAP-70 and TAP defects were ruled out. A missense mutation (gly90-->ser) in both alleles of the immunoglobulin domain of the CD8 alpha gene was shown to correlate with the absence of CD8 expression found in the patient and two sisters. Conversely, high percentages of CD4(-)CD8(-)TCR alpha beta(+) T cells were found in the three siblings. A novel autosomal recessive immunologic defect characterized by absence of CD8(+) cells is described. These findings may help to further understanding of the role of CD8 molecules in human immune response.

Publication types

  • Case Reports
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adult
  • Amino Acid Substitution*
  • Animals
  • Antibody Formation
  • Bacterial Infections / etiology
  • CD8 Antigens / chemistry
  • CD8 Antigens / genetics*
  • COS Cells
  • Chlorocebus aethiops
  • Consanguinity
  • Cytotoxicity, Immunologic
  • DNA Mutational Analysis
  • Dimerization
  • Female
  • Genes, Recessive
  • Genotype
  • Humans
  • Immunologic Deficiency Syndromes / genetics*
  • Immunologic Deficiency Syndromes / immunology
  • Immunologic Deficiency Syndromes / pathology
  • Male
  • Molecular Sequence Data
  • Mutation
  • Mutation, Missense*
  • Pedigree
  • Protein Subunits
  • Recombinant Fusion Proteins / immunology
  • Recurrence
  • Roma / genetics
  • Spain
  • T-Lymphocyte Subsets / immunology
  • T-Lymphocyte Subsets / pathology
  • Transfection

Substances

  • CD8 Antigens
  • Protein Subunits
  • Recombinant Fusion Proteins

Associated data

  • GENBANK/AY039664