Thyroid-infiltrating B lymphocytes in Graves' disease are related to marginal zone and memory B cell compartments

Thyroid. 2001 Jun;11(6):525-30. doi: 10.1089/105072501750302813.

Abstract

B lymphocytes that infiltrate the thyroid (Thy-B cells) in Graves' patients appear to be implicated in the pathophysiology of this disorder. The goal of the present study was to examine the nature of these Thy-B cells. To this end, Thy-B lymphocytes were isolated from surgical thyroidal samples, and their phenotype was determined by using mouse monoclonal antibodies (mAb) directed against a wide variety of surface markers, followed by flow cytometry multicolor analysis. The results show that most Thy-B cells (approximately 60%) exhibited IgM(+) IgD(low to -) surface immunoglobulin (Ig) profile, whereas the minor cell fraction (approximately 30%) consisted of switched IgG(+) memory B lymphocytes. Thy-B cells expressed low levels of CD5, CD23, and CD62L, which distinguished them from the resting B-cell pool, the major B-cell subset in the blood. In addition, they lacked CD38, CD10, and CD71, characteristic molecules for the germinal center B lymphocytes. In addition, Thy-B lymphocytes showed peculiar patterns both of adhesion molecules (CD62L(-), CD44(intermediate)), and of activation molecules (CD69(+), CD80(+), and, in part, CD95(+)). Taken together, these results suggest that the Thy-B lymphocyte subset consists of a combination of IgM(+) B cells resembling marginal zone B lymphocytes, and isotype-switched memory B cells.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adolescent
  • Adult
  • Antigens, CD / metabolism
  • B-Lymphocyte Subsets / physiology
  • B-Lymphocytes / pathology
  • B-Lymphocytes / physiology*
  • Biomarkers
  • Cell Adhesion Molecules / metabolism
  • Female
  • Flow Cytometry
  • Graves Disease / pathology
  • Graves Disease / physiopathology*
  • Humans
  • Immunohistochemistry
  • Immunologic Memory / physiology*
  • Phenotype
  • Thyroid Gland / pathology
  • Thyroid Gland / physiopathology*

Substances

  • Antigens, CD
  • Biomarkers
  • Cell Adhesion Molecules