Genes and mechanisms related to RNA interference regulate expression of the small temporal RNAs that control C. elegans developmental timing

Cell. 2001 Jul 13;106(1):23-34. doi: 10.1016/s0092-8674(01)00431-7.

Abstract

RNAi is a gene-silencing phenomenon triggered by double-stranded (ds) RNA and involves the generation of 21 to 26 nt RNA segments that guide mRNA destruction. In Caenorhabditis elegans, lin-4 and let-7 encode small temporal RNAs (stRNAs) of 22 nt that regulate stage-specific development. Here we show that inactivation of genes related to RNAi pathway genes, a homolog of Drosophila Dicer (dcr-1), and two homologs of rde-1 (alg-1 and alg-2), cause heterochronic phenotypes similar to lin-4 and let-7 mutations. Further we show that dcr-1, alg-1, and alg-2 are necessary for the maturation and activity of the lin-4 and let-7 stRNAs. Our findings suggest that a common processing machinery generates guide RNAs that mediate both RNAi and endogenous gene regulation.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Animals
  • Animals, Genetically Modified
  • Caenorhabditis elegans / embryology
  • Caenorhabditis elegans / genetics*
  • Caenorhabditis elegans / growth & development
  • DNA Primers
  • Drosophila / genetics
  • Embryo, Nonmammalian / physiology
  • Female
  • Gene Expression Regulation, Developmental*
  • Gene Silencing
  • Genes, Helminth
  • Genes, Reporter
  • Genomic Imprinting
  • Heterozygote
  • Larva
  • Luciferases / genetics
  • Phylogeny*
  • Polymerase Chain Reaction
  • RNA, Helminth / genetics*

Substances

  • DNA Primers
  • RNA, Helminth
  • Luciferases