Laxative-induced hypokalemia, sodium depletion and hyperreninemia. Effects of potassium and sodium replacement on the renin-angiotensin-aldosterone system

Ann Intern Med. 1975 Jul;83(1):60-2. doi: 10.7326/0003-4819-83-1-60.

Abstract

A patients with marked chronic hypokalemia (potassium, 1.7 to 2;3 meg/litre) and sodium depletion secondary to lazative abuse and dietary inadequacy was studied with respect to the renin-aldosterone system during sequential potassium and potassium-plus-sodium replacement. Extreme hyperreninemia of 20 Goldblatt units X 10-minus 4 was reduced to 0.9 with potassium replacement alone. Aldosteron excretion (15.8 mug/24 h) was initially low for a sodium-deprived state and high for a potassium-deprived state; it increased with potassium administration, but this rise was opposed by decreases in renin secretion induced by potassium and sodium administration. The results provide clinical confirmation of a dual effect of potassium on aldosterone secretion, with renin as a mediator.

MeSH terms

  • Aldosterone / metabolism*
  • Aldosterone / urine
  • Angiotensin II / metabolism*
  • Angiotensinogen / blood
  • Cathartics / poisoning*
  • Dose-Response Relationship, Drug
  • Female
  • Humans
  • Hypokalemia / chemically induced*
  • Hypokalemia / metabolism
  • Middle Aged
  • Potassium / metabolism*
  • Potassium / pharmacology
  • Radioimmunoassay
  • Renin / blood*
  • Renin / metabolism
  • Sodium / deficiency
  • Sodium / metabolism*
  • Sodium / pharmacology
  • Substance-Related Disorders
  • Water-Electrolyte Balance / drug effects

Substances

  • Cathartics
  • Angiotensinogen
  • Angiotensin II
  • Aldosterone
  • Sodium
  • Renin
  • Potassium