Aspirin inhibits NF-kappaB and protects from angiotensin II-induced organ damage

FASEB J. 2001 Aug;15(10):1822-4. doi: 10.1096/fj.00-0843fje.
No abstract available

MeSH terms

  • Angiotensin II*
  • Angiotensinogen / genetics
  • Animals
  • Animals, Genetically Modified
  • Aspirin / administration & dosage
  • Aspirin / pharmacology*
  • Aspirin / therapeutic use
  • CD4-Positive T-Lymphocytes / pathology
  • CD8-Positive T-Lymphocytes / pathology
  • Gene Expression
  • Heart Diseases / chemically induced
  • Heart Diseases / prevention & control*
  • Humans
  • I-kappa B Kinase
  • Kidney / blood supply
  • Kidney / chemistry
  • Kidney Diseases / chemically induced
  • Kidney Diseases / prevention & control*
  • Macrophages / pathology
  • Monocytes / pathology
  • Myocarditis / pathology
  • Myocarditis / prevention & control
  • Myocardium / chemistry
  • NF-kappa B / antagonists & inhibitors*
  • Nephritis / pathology
  • Nephritis / prevention & control
  • Protein Serine-Threonine Kinases / antagonists & inhibitors
  • Rats
  • Renin / genetics
  • Transcription Factor AP-1 / antagonists & inhibitors
  • Vascular Cell Adhesion Molecule-1 / analysis

Substances

  • NF-kappa B
  • Transcription Factor AP-1
  • Vascular Cell Adhesion Molecule-1
  • Angiotensinogen
  • Angiotensin II
  • Protein Serine-Threonine Kinases
  • CHUK protein, human
  • I-kappa B Kinase
  • IKBKB protein, human
  • IKBKE protein, human
  • Renin
  • Aspirin