Effect of photodynamic therapy (PDT) on the expression of pro-apoptotic protein Bak in nasopharyngeal carcinoma (NPC)

Lasers Surg Med. 2001;29(1):27-32. doi: 10.1002/lsm.1082.

Abstract

Background and objective: To investigate the effect of photodynamic therapy (PDT) on expression of the pro-apoptotic gene Bak in nasopharyngeal carcinoma (NPC).

Study design/materials and methods: Apoptosis and expression of the pro-apoptotic gene Bak on the tumor tissues from both pre- and post-PDT were determined using the in situ end labeling (ISEL), standard immunohistochemistry technique and western blot, respectively, in 24 patients with either persistent or recurrent NPC after radiotherapy.

Results: Before PDT, apoptotic index (AI) in tumor tissue was 1.2 +/- 0.6. At 6, 12, 24 and 48 hours after PDT, AI were 6.5 +/- 3.1, 23.6 +/- 8.3, 67.2 +/- 14.2 and 89.3 +/- 8.1, respectively. PDT caused apoptosis in a time-dependent fashion. Immunohistochemical assay indicated that 75% (18/24) of the patients had an upgrade expression of Bak protein in their tumor tissues after PDT. Increases in expression of Bak from PDT were also confirmed by western blot analysis.

Conclusions: PDT probably causes NPC cell apoptosis through an upregulation of the pro-apoptotic protein Bak expression.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adult
  • Aged
  • Apoptosis*
  • Blotting, Western
  • Carcinoma, Squamous Cell / drug therapy*
  • Carcinoma, Squamous Cell / metabolism*
  • Female
  • Hematoporphyrins / therapeutic use*
  • Humans
  • Immunohistochemistry
  • Male
  • Membrane Proteins / biosynthesis*
  • Middle Aged
  • Nasopharyngeal Neoplasms / drug therapy*
  • Nasopharyngeal Neoplasms / metabolism*
  • Photochemotherapy*
  • Radiation-Sensitizing Agents / therapeutic use*
  • Up-Regulation
  • bcl-2 Homologous Antagonist-Killer Protein

Substances

  • BAK1 protein, human
  • Hematoporphyrins
  • Membrane Proteins
  • Radiation-Sensitizing Agents
  • bcl-2 Homologous Antagonist-Killer Protein
  • photocarcinorin