Th2 dominance in nasal mucosa in patients with Wegener's granulomatosis

Clin Exp Immunol. 2001 Aug;125(2):332-9. doi: 10.1046/j.1365-2249.2001.125002332.x.

Abstract

Wegener's granulomatosis initially affects upper respiratory tract organs including the nasal mucosa in more than 90% of patients. The inflammation is typically granulomatous with associated vasculitis. T lymphocytes are usually a prominent component of the leucocyte infiltrate. Previous studies using peripheral blood T cells have implicated IFN-gamma rich Th1-type responses. This study addressed the cytokine milieu in nasal mucosa from 10 patients with active Wegener's granulomatosis using immunohistochemistry. Increased levels of CD3+ T cells and eosinophils were present compared with normal and disease controls. There was increased expression of IL-4, down-regulation of IL-2 and no detectable IFN-gamma. There was increased expression of the chemokine receptor CCR3 by infiltrating cells, consistent with an IL-4 dominant, Th2-biased response. In contrast, renal biopsy tissue from 10 patients with active Wegener's granulomatosis showed expression of IL-2 and IL-4. The Th2-type environment within nasal mucosa, often the initial site of disease activity in Wegener's, is consistent with a local allergic response in these patients.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adolescent
  • Adult
  • Aged
  • Biopsy
  • CD3 Complex / analysis
  • CD3 Complex / immunology
  • Cells, Cultured
  • Cytokines / biosynthesis
  • Cytokines / genetics
  • Cytokines / immunology
  • Female
  • Granulomatosis with Polyangiitis / immunology*
  • Granulomatosis with Polyangiitis / pathology
  • Humans
  • Immunity, Mucosal*
  • Immunohistochemistry
  • Kidney / immunology
  • Kidney / pathology
  • Male
  • Middle Aged
  • Myeloblastin
  • Nasal Mucosa / immunology*
  • Nasal Mucosa / pathology
  • RNA, Messenger / biosynthesis
  • Serine Endopeptidases / pharmacology
  • Th1 Cells / drug effects
  • Th1 Cells / immunology
  • Th2 Cells / drug effects
  • Th2 Cells / immunology*

Substances

  • CD3 Complex
  • Cytokines
  • RNA, Messenger
  • Serine Endopeptidases
  • Myeloblastin