Abstract
We used Y(1)R/LacZ transgenic mice to investigate the interaction between NPY, GABA and Y(1) receptors in the amygdala. Immunolabeling of GABA and NPY positive neurons and histochemical staining of beta-galactosidase revealed NPY and GABA colocalization and close contacts of NPY-positive fibers with GABAergic neurons also expressing the Y(1)R/LacZ transgene.
Publication types
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Research Support, Non-U.S. Gov't
MeSH terms
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Amygdala / anatomy & histology*
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Amygdala / metabolism*
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Animals
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Gene Expression / physiology
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Male
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Mice
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Mice, Transgenic
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Nerve Net / metabolism
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Neuropeptide Y / metabolism
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Neuropeptide Y / physiology*
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Receptors, GABA / metabolism
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Receptors, GABA / physiology*
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Receptors, Neuropeptide Y / metabolism
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Receptors, Neuropeptide Y / physiology*
Substances
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Neuropeptide Y
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Receptors, GABA
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Receptors, Neuropeptide Y
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neuropeptide Y-Y1 receptor