Leukocyte integrin Mac-1 recruits toll/interleukin-1 receptor superfamily signaling intermediates to modulate NF-kappaB activity

Circ Res. 2001 Nov 9;89(10):859-65. doi: 10.1161/hh2201.099166.

Abstract

The leukocyte integrin Mac-1 (alphaMbeta2, CD11b/CD18) regulates important cell functions in inflammation, including adhesion, phagocytosis, and oxidative burst. Deficiency of Mac-1 reduces vessel wall inflammation and neointimal thickening after murine carotid artery injury. Although Mac-1 has been implicated in modulating AP-1 and NF-kappaB activity, the signal transduction pathways involved are undefined. cDNA array analysis of Mac-1-clustered compared with -nonclustered monocytic THP-1 cells showed increased expression of the signal transducer TRAF6 (TNF receptor-associated factor 6), leading us to consider the possibility that Mac-1 used a Toll/IL-1 receptor family-like signaling pathway. Mac-1-dependent activation of NF-kappaB was potentiated by wild-type, and attenuated by dominant negative, TRAF6- and TGF-beta-activated kinase (TAK1) constructs. IRAK1 (IL-1 receptor associated kinase), a kinase immediately upstream of TRAF6, coimmunoprecipitated with Mac-1. Taken together, these observations indicate that Mac-1 recruits a Toll/IL-1 receptor family-like cascade to modulate NF-kappaB activity. This represents a new pathway for integrin-dependent modulation of gene expression.

Publication types

  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Adaptor Proteins, Signal Transducing
  • Antigens, Differentiation / genetics
  • Antigens, Differentiation / metabolism
  • Cell Adhesion / immunology
  • Cell Line
  • Gene Expression Regulation / immunology
  • Genes, Reporter
  • Humans
  • Interleukin-1 Receptor-Associated Kinases
  • MAP Kinase Kinase Kinases / metabolism
  • Macrophage-1 Antigen / genetics
  • Macrophage-1 Antigen / metabolism*
  • Membrane Glycoproteins*
  • Membrane Proteins / metabolism*
  • Monocytes / cytology
  • Monocytes / immunology
  • Monocytes / metabolism
  • Myeloid Differentiation Factor 88
  • NF-kappa B / genetics
  • NF-kappa B / metabolism*
  • Oligonucleotide Array Sequence Analysis
  • Protein Kinases / metabolism
  • Proteins / metabolism
  • Receptor Aggregation / immunology
  • Receptors, Cell Surface*
  • Receptors, Immunologic / metabolism*
  • Receptors, Interleukin-1 / metabolism*
  • Signal Transduction / immunology
  • TNF Receptor-Associated Factor 6
  • Toll-Like Receptors
  • Transfection

Substances

  • Adaptor Proteins, Signal Transducing
  • Antigens, Differentiation
  • MYD88 protein, human
  • Macrophage-1 Antigen
  • Membrane Glycoproteins
  • Membrane Proteins
  • Myeloid Differentiation Factor 88
  • NF-kappa B
  • Proteins
  • Receptors, Cell Surface
  • Receptors, Immunologic
  • Receptors, Interleukin-1
  • TNF Receptor-Associated Factor 6
  • Toll-Like Receptors
  • Protein Kinases
  • IRAK1 protein, human
  • Interleukin-1 Receptor-Associated Kinases
  • MAP Kinase Kinase Kinases
  • MAP kinase kinase kinase 7