Secretory activity of bovine ovarian granulosa cells transfected with sense and antisense insulin-like growth factor (IGF) binding protein-3 and the response to IGF-I, GH, LH, oxytocin and oestradiol

J Mol Endocrinol. 2001 Dec;27(3):329-38. doi: 10.1677/jme.0.0270329.

Abstract

The aim of our in vitro experiments was to examine if IGF binding protein (IGFBP)-3 is involved in control of bovine ovarian secretory activity. For this purpose we performed the transfection of bovine granulosa cells with cDNA sense and antisense constructs increasing or inhibiting IGFBP-3 synthesis. The release of IGFBP-3, progesterone, oxytocin, IGF-I and prostaglandins F (PGF) and E (PGE) by control and transfected cells was compared. The transfected ovarian cells were cultured with and without bLH (100 ng/ml), bGH (100 ng/ml), IGF-I (10 ng/ml), oxytocin (10 ng/ml) and oestradiol-17beta (100 ng/ml). The concentration of IGFBP-3 produced was assessed using ligand and western blotting and secretion of progesterone, oxytocin, IGF-I, PGF and PGE was evaluated using RIA/IRMA techniques. Transfection of cells with the sense IGFBP-3 cDNA construct resulted in the expected increase in IGFBP-3 release, whereas the antisense IGFBP-3 construct induced the expected reduction in IGFBP-3 output. The granulosa cells transfected to overexpress IGFBP-3 had an increase in IGF-I, PGF and PGE release, and a decrease in basal and hormone- or growth factor-induced accumulation of progesterone and oxytocin. The granulosa cells transfected to have reduced IGFBP-3 expression gave primarily significant opposite findings. The present results suggest the involvement of IGFBP-3 in control of bovine ovarian steroid, peptide hormone, growth factor and prostaglandin release. IGFBP-3 is a physiological stimulator of IGF-I and prostaglandin release and an inhibitor of steroid and peptide hormone output.

MeSH terms

  • Animals
  • Cattle
  • DNA, Antisense / genetics
  • DNA, Complementary / genetics
  • Estradiol / pharmacology
  • Female
  • Granulosa Cells / drug effects*
  • Granulosa Cells / metabolism*
  • Growth Hormone / pharmacology
  • Hormones / pharmacology*
  • In Vitro Techniques
  • Insulin-Like Growth Factor Binding Protein 3 / genetics*
  • Insulin-Like Growth Factor Binding Protein 3 / metabolism
  • Insulin-Like Growth Factor I / metabolism
  • Insulin-Like Growth Factor I / pharmacology
  • Luteinizing Hormone / pharmacology
  • Oxytocin / metabolism
  • Oxytocin / pharmacology
  • Progesterone / metabolism
  • Prostaglandins E / metabolism
  • Prostaglandins F / metabolism
  • Recombinant Proteins / genetics
  • Recombinant Proteins / metabolism
  • Transfection

Substances

  • DNA, Antisense
  • DNA, Complementary
  • Hormones
  • Insulin-Like Growth Factor Binding Protein 3
  • Prostaglandins E
  • Prostaglandins F
  • Recombinant Proteins
  • Progesterone
  • Estradiol
  • Oxytocin
  • Insulin-Like Growth Factor I
  • Luteinizing Hormone
  • Growth Hormone