Hypercholesterolemia alters vascular functions and gene expression of potassium channels in rat aortic smooth muscle cells

Acta Pharmacol Sin. 2001 Mar;22(3):274-8.

Abstract

Aim: To investigate the effects of hypercholesterolemia on functions of rat aorta and on gene expression of inward rectifier and ATP-sensitive potassium channels in aortic smooth muscle.

Methods: Rats were treated with high-cholesterol emulsion ig for 2 weeks; the aortic rings with and without endothelium were prepared to examine the aortic contractile and relaxation responses; RT-PCR was used to observe the gene expression of inward rectifier and ATP-sensitive potassium channels.

Results: Hypercholesterolemia damaged the endothelium-dependent vascular functions severely, but did not affect the endothelium-independent vascular functions; Kir6.2 mRNA expression was upregulated (P < 0.05) and Kir3.1 mRNA expression was downregulated markedly (P < 0.05) in hypercholesterolemic smooth muscle.

Conclusion: Hypercholesterolemia altered the vascular functions and regulated gene expression levels of specific inward rectifier and ATP-sensitive potassium channel subtypes.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Aorta, Thoracic / metabolism
  • Aorta, Thoracic / pathology
  • Gene Expression
  • Hypercholesterolemia / metabolism*
  • Hypercholesterolemia / physiopathology
  • Male
  • Muscle Contraction / physiology
  • Muscle, Smooth, Vascular / metabolism*
  • Muscle, Smooth, Vascular / pathology
  • Potassium Channels / biosynthesis*
  • Potassium Channels / genetics
  • Potassium Channels, Inwardly Rectifying / biosynthesis*
  • Potassium Channels, Inwardly Rectifying / genetics
  • RNA, Messenger / genetics
  • Rats
  • Rats, Wistar

Substances

  • Potassium Channels
  • Potassium Channels, Inwardly Rectifying
  • RNA, Messenger