Inhibition of both skin and lung tumorigenesis by Car-R mouse-derived cancer modifier loci

Int J Cancer. 2002 Feb 10;97(5):580-3. doi: 10.1002/ijc.10104.

Abstract

The Car-R outbred mouse line was phenotypically selected for high resistance to two-stage skin tumorigenesis. In the present study we tested the hypothesis that a subset of genetic loci responsible for resistance to skin tumorigenesis of Car-R mice might also inhibit lung tumorigenesis. Skin and lung tumorigenesis were induced in groups of Car-R, SWR/J, (SWR/JxCar-R)F1 and SWR/Jx(SWR/JxCar-R) backcross mice by i.p. urethane initiation and skin TPA promotion. Car-R mice showed a much lower susceptibility to both skin and lung tumorigenesis as compared to SWR/J mice, which are susceptible to both lung and skin tumorigenesis. The Car-R-inherited genome significantly inhibited both skin and lung cancer development in the F1 progeny of Car-R with SWR/J mice. In the backcross population, skin and lung tumor phenotypes showed a statistically significant correlation, indicating that a subset of the cancer resistance alleles, which segregated in the Car-R line during selection for resistance to skin carcinogenesis, provides resistance to both skin and lung tumorigenesis.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Alleles
  • Animals
  • Animals, Outbred Strains
  • Carcinogenicity Tests
  • Carcinogens
  • Female
  • Genetic Predisposition to Disease*
  • Inbreeding
  • Incidence
  • Lung Neoplasms / chemically induced
  • Lung Neoplasms / epidemiology
  • Lung Neoplasms / genetics*
  • Male
  • Mice
  • Mice, Inbred Strains
  • Neoplasms, Experimental / chemically induced
  • Neoplasms, Experimental / epidemiology
  • Neoplasms, Experimental / genetics*
  • Papilloma / chemically induced
  • Papilloma / epidemiology
  • Papilloma / genetics*
  • Phenotype
  • Polymerase Chain Reaction
  • Polymorphism, Genetic
  • Proto-Oncogene Proteins / genetics
  • Proto-Oncogene Proteins p21(ras)
  • Skin Neoplasms / chemically induced
  • Skin Neoplasms / epidemiology
  • Skin Neoplasms / genetics*
  • Tetradecanoylphorbol Acetate
  • Urethane
  • ras Proteins

Substances

  • Carcinogens
  • KRAS protein, human
  • Proto-Oncogene Proteins
  • Urethane
  • Proto-Oncogene Proteins p21(ras)
  • ras Proteins
  • Tetradecanoylphorbol Acetate