Prenatal high protein exposure decreases energy expenditure and increases adiposity in young rats

J Nutr. 2002 Feb;132(2):142-4. doi: 10.1093/jn/132.2.142.

Abstract

Epidemiologic results suggest that protein intake in infancy and later adiposity might be related. We examined whether high dietary protein exposure in utero and/or during postnatal life affects body fatness. Two groups of female rats were mated and pair-fed isocaloric high (40% protein; HP) or adequate protein (20% protein; AP) diets throughout pregnancy. The male offspring were suckled (3 wk) by foster mothers pair-fed HP or AP diets, resulting in 4 pre-/postnatal groups (AP-AP, AP-HP, HP-AP, HP-HP). Subsequently, they were pair-fed the same diets their nurses received during lactation until wk 9. Offspring of HP dams had a lower body weight on d 2 of life than their AP counterparts (7.6 +/- 0.7 vs. 8.3 +/- 0.8 g; P < 0.001). HP-AP rats had a higher body weight than AP-AP controls at wk 3, 5, and 6 (P < 0.05), in contrast to HP-HP which did not differ from controls. Prenatal HP exposure resulted in a greater total and relative fat mass and decreased total energy expenditure at wk 9 (P < 0.05). Postnatal HP alone had no significant effect on body composition or metabolic rate. These results indicate that in utero exposure to a high protein level reprograms body weight and energy homeostasis.

MeSH terms

  • Adipose Tissue / drug effects*
  • Adipose Tissue / growth & development
  • Aging / physiology*
  • Animals
  • Body Composition
  • Dietary Proteins / administration & dosage*
  • Dietary Proteins / metabolism
  • Energy Metabolism / drug effects*
  • Energy Metabolism / physiology
  • Female
  • Growth / physiology
  • Male
  • Nutritional Physiological Phenomena
  • Obesity / etiology*
  • Obesity / physiopathology
  • Pregnancy
  • Prenatal Exposure Delayed Effects*
  • Rats
  • Rats, Wistar

Substances

  • Dietary Proteins