Degeneration and restoration of spermatogenesis in relation to the changes in Leydig cell population following ethane dimethanesulfonate treatment in adult rats

Endocr Regul. 2001 Dec;35(4):209-15.

Abstract

Objective: To investigate degeneration and restoration patterns of spermatogenesis in relation to the changes in Leydig cells (LCs) after treatment with ethane dimethanesulfonate (EDS).

Materials and methods: Adult Wistar male rats were treated with EDS at a dose 75 mg/kg body weight and the testes were sampled at 7, 14, 21, 35 and 49 days after treatment for histological and ultrastructural studies.

Results: During the first two weeks after treatment stage dependent loss of germ cells was found within seminiferous tubules that led to a profound disturbance of spermatogenesis. The restoration of seminiferous epithelium followed also in stage specific manner and in relation to development of a new LC population (third week). The development of new LCs after EDS treatment repeats the normal dynamics of postnatal LC development within a similar time range.

Conclusion: EDS treatment of rats causes a temporary germ cell degeneration in the testis. The kinetics of disappearance of germ cells and their regeneration broadly follows the changes in LC population.

MeSH terms

  • Animals
  • Cell Count
  • Follicle Stimulating Hormone / metabolism
  • Kinetics
  • Leydig Cells / drug effects*
  • Luteinizing Hormone / metabolism
  • Male
  • Mesylates / pharmacology*
  • Microscopy, Electron
  • Pituitary Gland / metabolism
  • Rats
  • Rats, Wistar
  • Seminiferous Epithelium / cytology
  • Seminiferous Epithelium / drug effects
  • Spermatogenesis / drug effects*
  • Testis / metabolism
  • Testosterone / blood
  • Testosterone / metabolism

Substances

  • Mesylates
  • Testosterone
  • Luteinizing Hormone
  • Follicle Stimulating Hormone
  • ethylene dimethanesulfonate