A novel mutation of the tyrosinase gene causing oculocutaneous albinism type 1 (OCA1)

J Dermatol Sci. 2002 Feb;28(2):102-5. doi: 10.1016/s0923-1811(01)00141-4.

Abstract

Tyrosinase is a rate-limiting enzyme in the melanin biosynthetic pathway and a complete defect of the enzyme activity caused by homozygous mutations of the tyrosinase gene is well known to result in tyrosinase-negative oculocutaneous albinism (OCA1A) patients who never develop any melanin pigment in the skin, hair and eyes throughout life. In this paper, we report a novel missense substitution, R239W(CGG --> TGG) of the tyrosinase gene in a patient with tyrosinase-negative OCA.

Publication types

  • Case Reports
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Albinism, Oculocutaneous / enzymology*
  • Albinism, Oculocutaneous / genetics*
  • Amino Acid Sequence
  • Base Sequence
  • DNA / genetics
  • Female
  • Frameshift Mutation
  • Heterozygote
  • Humans
  • Infant
  • Male
  • Monophenol Monooxygenase / deficiency*
  • Monophenol Monooxygenase / genetics*
  • Mutation, Missense*
  • Pedigree

Substances

  • DNA
  • Monophenol Monooxygenase