Inhibition of heat shock-induced apoptosis by peppermint oil in astrocytes

J Mol Neurosci. 2001 Dec;17(3):391-6. doi: 10.1385/JMN:17:3:391.

Abstract

Exposure to environmental stresses and toxins is linked to the pathogenesis of neuropsychiatric disorders. Astrocytes, the most abundant glial-cell type in the brain, are considered to have physiological and pathological roles in neuronal activities. We have investigated whether peppermint oil inhibits heat shock-induced apoptosis of astrocytes. We found that peppermint oil inhibits the heat shock-induced apoptosis in both human astrocyte CCF-STTG1 cells and rat astrocytes. Pretreatment of the cells with peppermint oil inhibited the heat shock-induced DNA fragmentation and condensation of nuclear chromatin. Peppermint oil also inhibited the caspase-3 activation and poly-ADP-ribose polymerase fragmentation in CCF-STTG1 cells. These results suggest that peppermint oil may modulate the apoptosis of astrocytes via the activation of the caspase-3.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Apoptosis / drug effects*
  • Astrocytes / drug effects
  • Astrocytes / metabolism*
  • Blotting, Western
  • Caspase 3
  • Caspases / drug effects
  • Caspases / metabolism
  • Cell Culture Techniques
  • DNA Fragmentation / drug effects
  • Electrophoresis
  • Enzyme Activation / drug effects
  • Flow Cytometry
  • Hot Temperature / adverse effects*
  • Humans
  • Mentha piperita
  • Plant Oils / pharmacology*
  • Poly (ADP-Ribose) Polymerase-1
  • Poly(ADP-ribose) Polymerases
  • Proteins / drug effects
  • Proteins / metabolism
  • Rats

Substances

  • Plant Oils
  • Proteins
  • peppermint oil
  • PARP1 protein, human
  • Parp1 protein, rat
  • Poly (ADP-Ribose) Polymerase-1
  • Poly(ADP-ribose) Polymerases
  • CASP3 protein, human
  • Casp3 protein, rat
  • Caspase 3
  • Caspases