Extensive in vivo self-renewal, long-term reconstitution capacity, and hematopoietic multipotency of Pax5-deficient precursor B-cell clones

Blood. 2002 Apr 15;99(8):2760-6. doi: 10.1182/blood.v99.8.2760.

Abstract

Self-renewal, pluripotency, and long-term reconstitution are defining characteristics of single hematopoietic stem cells. Pax5(-/-) precursor B cells apparently possess similar characteristics. Here, using serial transplantations, with in vitro recloning and growth of the bone marrow-homed donor cells occurring after all transplantations, we analyzed the extent of self-renewal and hematopoietic multipotency of Pax5(-/-) precursor B-cell clones. Moreover, telomere length and telomerase activity in these clones was analyzed at various time points. Thus far, 5 successive transplantations have been performed. Clones transplanted for the fifth time, which have proliferated for more than 150 cell divisions in vitro, still repopulate the bone marrow with precursor B cells and reconstitute these recipients with lymphoid and myeloid cells. During this extensive proliferation, Pax5(-/-) precursor B cells shorten their telomeres at 70 to 90 base pairs per division. Their telomerase activity remains at 3% of that of HEK293 cancer cells during all serial in vivo transplantations/in vitro expansions. Together, these data show that Pax5(-/-) precursor B-cell clones possess extensive in vivo self-renewal capacity, long-term reconstitution capacity, and hematopoietic multipotency, with their telomeres shortening at the normal rate.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • B-Lymphocytes / cytology*
  • B-Lymphocytes / physiology
  • Bone Marrow
  • Cell Differentiation
  • Cell Division
  • Cell Movement / physiology*
  • Clone Cells / cytology
  • Clone Cells / physiology
  • Clone Cells / transplantation
  • DNA-Binding Proteins / deficiency
  • DNA-Binding Proteins / genetics
  • Hematopoiesis*
  • Hematopoietic Stem Cell Transplantation*
  • Hematopoietic Stem Cells / cytology*
  • Hematopoietic Stem Cells / physiology
  • Mice
  • Mice, Knockout
  • PAX5 Transcription Factor
  • Telomere / metabolism
  • Telomere / ultrastructure
  • Transcription Factors / deficiency
  • Transcription Factors / genetics

Substances

  • DNA-Binding Proteins
  • PAX5 Transcription Factor
  • Pax5 protein, mouse
  • Transcription Factors