B7-CTLA4 interaction promotes cognate destruction of tumor cells by cytotoxic T lymphocytes in vivo

Blood. 2002 Apr 15;99(8):2880-9. doi: 10.1182/blood.v99.8.2880.

Abstract

Costimulatory molecules B7-1 and B7-2 (hereby collectively called B7) interact with CD28 and CTLA4 on T cells and promote antitumor immunity. The function of B7-CTLA4 interaction in antitumor CTL response remains controversial. Here we used CD28(-/-) and CD28(+/-) or CD28(+/+) transgenic mice that express the T-cell receptor specific for an unmutated tumor antigen, P1A, and for tumor cells expressing a CTLA4-specific B7 mutant to evaluate the function of CD28-B7 and CTLA4-B7 interactions in induction and effector phases of antitumor immunity. We report that B7-CD28 and B7-CTLA4 interactions promote tumor rejection. However, this is achieved by distinct mechanisms. B7-CD28 interaction enhances T-cell clonal expansion, though a role for this interaction in the effector phase cannot be ruled out. In contrast, B7-CTLA4 interaction enhances the CTL-mediated destruction of tumors, but not T-cell clonal expansion.

Publication types

  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Abatacept
  • Adoptive Transfer
  • Animals
  • Antigens, CD / genetics
  • Antigens, CD / immunology
  • Antigens, CD / metabolism
  • Antigens, Differentiation / immunology
  • Antigens, Differentiation / metabolism*
  • Antigens, Neoplasm / immunology*
  • B7-1 Antigen / genetics
  • B7-1 Antigen / immunology
  • B7-1 Antigen / metabolism*
  • B7-2 Antigen
  • CD28 Antigens / genetics
  • CD28 Antigens / metabolism
  • CTLA-4 Antigen
  • Cytotoxicity, Immunologic / drug effects
  • Immunity
  • Immunoconjugates*
  • Lymphocyte Activation / drug effects
  • Membrane Glycoproteins / genetics
  • Membrane Glycoproteins / immunology
  • Membrane Glycoproteins / metabolism
  • Mice
  • Mice, Transgenic
  • Mutation
  • Neoplasms, Experimental / therapy
  • Protein Binding / immunology
  • Receptors, Antigen, T-Cell / immunology
  • T-Lymphocytes, Cytotoxic / immunology*
  • T-Lymphocytes, Cytotoxic / transplantation

Substances

  • Antigens, CD
  • Antigens, Differentiation
  • Antigens, Neoplasm
  • B7-1 Antigen
  • B7-2 Antigen
  • CD28 Antigens
  • CTLA-4 Antigen
  • Cd86 protein, mouse
  • Ctla4 protein, mouse
  • Immunoconjugates
  • Membrane Glycoproteins
  • Receptors, Antigen, T-Cell
  • tumor rejection antigen P815A, mouse
  • Abatacept