Elevated monocytic IL-12 and TNF-alpha production in Wegener's granulomatosis is normalized by cyclophosphamide and corticosteroid therapy

Clin Exp Immunol. 2002 Apr;128(1):181-6. doi: 10.1046/j.1365-2249.2002.01801.x.

Abstract

Wegener's granulomatosis (WG) is characterized by a predominance of the type 1 T-helper cell (Th1) response. We have studied monocytic cytokine expression in untreated patients and in patients who did not respond to prior methotrexate or trimethoprim-sulphamethoxazole therapy, i.e. patients with active disease. Intracytoplasmic IL-12 and TNF-alpha expression was significantly increased in WG compared with healthy controls. IL-8 expression was not increased. Two and 12 weeks of daily standard oral cyclophosphamide and corticosteroid (CYC + GC) treatment induced a stable remission of the disease. Elevated IL-12 and TNF-alpha expression of monocytes was normalized. The active metabolite of CYC was shown to down-regulate IL-12 mRNA in vitro. Monocytic cytokines, especially IL-12, may have a role in the early determination and skewing of the immunoregulatory response towards a Th1 profile. It appears that CYC + GC exerts its effect by normalizing the Th1-driving cytokine pattern, and CYC may maintain this mode of action. Normalization of the skewed cytokine pattern may be a prerequisite and an indicator of inducing a remission in WG.

Publication types

  • Clinical Trial
  • Comparative Study
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adrenal Cortex Hormones / therapeutic use*
  • Adult
  • Cells, Cultured
  • Cyclophosphamide / pharmacology
  • Cyclophosphamide / therapeutic use*
  • Cytokines / biosynthesis*
  • Cytoplasm / metabolism
  • Down-Regulation
  • Female
  • Granulomatosis with Polyangiitis / drug therapy*
  • Granulomatosis with Polyangiitis / immunology*
  • Humans
  • Interleukin-12 / biosynthesis
  • Interleukin-12 / genetics
  • Interleukin-8 / metabolism
  • Kinetics
  • Longitudinal Studies
  • Male
  • Middle Aged
  • Monocytes / drug effects
  • Monocytes / immunology*
  • RNA, Messenger / biosynthesis
  • Remission Induction
  • Treatment Outcome
  • Tumor Necrosis Factor-alpha / biosynthesis

Substances

  • Adrenal Cortex Hormones
  • Cytokines
  • Interleukin-8
  • RNA, Messenger
  • Tumor Necrosis Factor-alpha
  • Interleukin-12
  • Cyclophosphamide