Abstract
Listeria monocytogenes (Lm) is an attractive vector to elicit T cell immunity because it infects antigen-presenting cells and because infection originates at the mucosa. Lm expressing HIV gag elicits sustained high levels of gag-specific CTL in mice. Since Lm causes disease in immunocompromised hosts, a highly attenuated strain of Lm that requires D-Ala for viability was produced. Attenuated bacteria expressing HIV-1 gag (Lmdd-gag) are as efficient as wild-type recombinants at stimulating gag-specific murine CTL when administered with D-Ala and at boosting human CTL in vitro. Lmdd-gag immunization protects mice from vaccinia-gag challenge and induces mucosal CTL, even after systemic immunization.
MeSH terms
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AIDS Vaccines* / genetics
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AIDS Vaccines* / immunology
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AIDS Vaccines* / toxicity
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Adult
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Animals
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Bacterial Vaccines / immunology
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Bacterial Vaccines / toxicity
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CD4-Positive T-Lymphocytes / immunology
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CD8-Positive T-Lymphocytes / immunology
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Female
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Genes, gag*
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Genes, nef
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Genetic Engineering
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Genetic Vectors / genetics
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Genetic Vectors / immunology*
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HIV-1 / genetics
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HIV-1 / immunology*
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Humans
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Immunity, Mucosal
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Infant, Newborn
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Listeria monocytogenes / genetics
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Listeria monocytogenes / immunology*
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Male
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Mice
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Pregnancy
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Safety
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T-Lymphocytes, Cytotoxic / immunology
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Vaccines, Attenuated / immunology
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Vaccines, Synthetic / genetics
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Vaccines, Synthetic / immunology
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Vaccines, Synthetic / toxicity
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Vaccinia virus / immunology
Substances
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AIDS Vaccines
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Bacterial Vaccines
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Vaccines, Attenuated
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Vaccines, Synthetic