Abstract
Electrical silencing of Drosophila circadian pacemaker neurons through targeted expression of K+ channels causes severe deficits in free-running circadian locomotor rhythmicity in complete darkness. Pacemaker electrical silencing also stops the free-running oscillation of PERIOD (PER) and TIMELESS (TIM) proteins that constitutes the core of the cell-autonomous molecular clock. In contrast, electrical silencing fails to abolish PER and TIM oscillation in light-dark cycles, although it does impair rhythmic behavior. On the basis of these findings, we propose that electrical activity is an essential element of the free-running molecular clock of pacemaker neurons along with the transcription factors and regulatory enzymes that have been previously identified as required for clock function.
Publication types
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Research Support, Non-U.S. Gov't
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Research Support, U.S. Gov't, Non-P.H.S.
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Research Support, U.S. Gov't, P.H.S.
MeSH terms
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Action Potentials / genetics*
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Animals
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Biological Clocks / genetics*
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Cell Size / genetics
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Circadian Rhythm / genetics*
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Dark Adaptation / genetics
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Drosophila Proteins*
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Drosophila melanogaster / cytology
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Drosophila melanogaster / genetics
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Drosophila melanogaster / metabolism*
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Gene Expression Regulation, Developmental / genetics
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Genes, Lethal / genetics
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Insect Proteins / genetics
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Insect Proteins / metabolism
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Motor Activity / genetics
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Mutation / genetics
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Nervous System / cytology
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Nervous System / embryology
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Nervous System / metabolism*
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Neurons / cytology
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Neurons / metabolism*
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Nuclear Proteins / genetics
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Nuclear Proteins / metabolism
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Period Circadian Proteins
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Photic Stimulation
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Potassium Channels / deficiency*
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Potassium Channels / genetics
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Potassium Channels, Inwardly Rectifying / genetics
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Potassium Channels, Inwardly Rectifying / metabolism
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Synapses / genetics
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Synapses / metabolism
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Synaptic Transmission / genetics*
Substances
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Drosophila Proteins
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Insect Proteins
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Nuclear Proteins
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Ork1 protein, Drosophila
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PER protein, Drosophila
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Period Circadian Proteins
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Potassium Channels
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Potassium Channels, Inwardly Rectifying
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tim protein, Drosophila