G120K-PEG, a human GH antagonist, decreases GH signal transduction in the liver of mice

Mol Cell Endocrinol. 2002 Jun 28;192(1-2):65-74. doi: 10.1016/s0303-7207(02)00110-7.

Abstract

After receptor binding, growth hormone (GH) induces GH receptors (GHR) dimerization and JAK2 is activated after its association with a dimerized GHR, stimulating the tyrosyl phosphorylation of insulin receptor substrate-1 (IRS-1), IRS-2 and Shc proteins. G120K-PEG, a GH antagonist is produced by a mutation that blocks GH action by preventing the GHR dimerization. This study shows that the inhibitory effect of G120K-PEG was maximal with a GH:G120K-PEG ratio of 1:100, as no increase in JAK2 tyrosyl phosphorylation was observed with this dose of GH. When the dose of GH was increased and with a GH:G120K-PEG ratio of 1:10 some tyrosyl phosphorylation of JAK2 could be observed. Additionally, GH-induced IRS-1, IRS-2 and SHC tyrosyl phosphorylation was inhibited approximately 50% at equimolar concentrations of the antagonist of GH and almost abolished with a GH:G120K-PEG ratio of 1:100. The results clearly show that G120K-PEG inhibits GH signal transduction in mouse liver.

MeSH terms

  • Adaptor Proteins, Signal Transducing*
  • Adaptor Proteins, Vesicular Transport*
  • Animals
  • Dimerization
  • Dose-Response Relationship, Drug
  • Hormone Antagonists / pharmacology*
  • Human Growth Hormone / antagonists & inhibitors*
  • Human Growth Hormone / pharmacology
  • Humans
  • Insulin Receptor Substrate Proteins
  • Intracellular Signaling Peptides and Proteins
  • Janus Kinase 2
  • Liver / drug effects*
  • Liver / metabolism
  • Male
  • Mice
  • Phosphoproteins / metabolism
  • Phosphorylation / drug effects
  • Phosphotyrosine / analysis
  • Polyethylene Glycols / pharmacology*
  • Prolactin / pharmacology
  • Protein Processing, Post-Translational / drug effects
  • Protein-Tyrosine Kinases / metabolism
  • Proteins / metabolism
  • Proto-Oncogene Proteins*
  • Receptors, Somatotropin / chemistry
  • Receptors, Somatotropin / drug effects
  • Shc Signaling Adaptor Proteins
  • Signal Transduction / drug effects*
  • Src Homology 2 Domain-Containing, Transforming Protein 1

Substances

  • Adaptor Proteins, Signal Transducing
  • Adaptor Proteins, Vesicular Transport
  • G120K-PEG
  • Hormone Antagonists
  • IRS1 protein, human
  • IRS2 protein, human
  • Insulin Receptor Substrate Proteins
  • Intracellular Signaling Peptides and Proteins
  • Irs1 protein, mouse
  • Irs2 protein, mouse
  • Phosphoproteins
  • Proteins
  • Proto-Oncogene Proteins
  • Receptors, Somatotropin
  • SHC1 protein, human
  • Shc Signaling Adaptor Proteins
  • Shc1 protein, mouse
  • Src Homology 2 Domain-Containing, Transforming Protein 1
  • Human Growth Hormone
  • Phosphotyrosine
  • Polyethylene Glycols
  • Prolactin
  • Protein-Tyrosine Kinases
  • JAK2 protein, human
  • Jak2 protein, mouse
  • Janus Kinase 2