Impact of laparoscopic CO2-insufflation on tumor-associated molecules in cultured colorectal cancer cells

Surg Endosc. 2002 Aug;16(8):1182-6. doi: 10.1007/s00464-001-9194-3. Epub 2002 Apr 9.

Abstract

Background: Laparoscopic CO2-insufflation is believed to stimulate proliferation and metastatic potential of gastrointestinal carcinomas. E-cadherin, I-CAM1, I-CAM2, and CD44 are involved into the metastatic process of different cancer cell lines. The current study investigates the influence of CO2-insufflation on the expression of E-cadherin, I-CAM1, I-CAM2, and CD44 in vitro.

Methods: CX-2 and CC531 colon carcinoma cells (human/rat) were exposed to pneumoperitoneal CO2-insufflation. E-cadherin, I-CAM1, I-CAM2, and CD44 were measured 0, 12, 24, 48, and 72 h after CO2-insufflation using flowcytometry. Control groups were exposed to room air. Data were analyzed by the Wilcoxon-Mann-Whitney U-test.

Results: Both cell lines showed significant alteration in E-cadherin, I-CAM1, and CD44 expression after CO2 exposure (p <0.05). No significant differences were found regarding I-CAM2 expression.

Conclusion: The present study demonstrates CO2-insufflation to influence the expression of E-cadherin, I-CAM1 and CD44. Whether these changes increases the metastatic potential of colorectal cancer cells in vivo needs further investigation.

MeSH terms

  • Adenocarcinoma / physiopathology*
  • Adenocarcinoma / secondary
  • Air
  • Animals
  • Cadherins / metabolism
  • Carbon Dioxide
  • Colonic Neoplasms / physiopathology*
  • Humans
  • Hyaluronan Receptors / metabolism
  • Insufflation / adverse effects*
  • Intercellular Adhesion Molecule-1 / metabolism
  • Laparoscopy / adverse effects*
  • Rats
  • Tumor Cells, Cultured

Substances

  • Cadherins
  • Hyaluronan Receptors
  • Intercellular Adhesion Molecule-1
  • Carbon Dioxide