Microparticle subpopulations are increased in preeclampsia: possible involvement in vascular dysfunction?

Am J Obstet Gynecol. 2002 Aug;187(2):450-6. doi: 10.1067/mob.2002.124279.

Abstract

Objective: The purpose of this study was to investigate the cellular origin and numbers of circulating microparticles in normal pregnancy and preeclampsia.

Study design: Plasma samples from 10 women with preeclampsia, from 10 normal pregnant women, and from 10 nonpregnant women matched for age and gestation, were analyzed by flow cytometry.

Results: The total number of circulating microparticles was unaltered in pregnancy and preeclampsia. The largest portion of microparticles was derived from platelets in all groups. T-suppressor cell microparticle numbers were decreased in normal pregnancy (P =.04). In preeclampsia T-suppressor, T-helper cell, and granulocyte microparticle numbers were increased (P =.008,.008, and.03, respectively). Elastase concentrations were increased in preeclampsia (P =.02) and correlated with granulocyte microparticle numbers (P =.006). Elastase concentrations correlated with systolic and diastolic blood pressure (P =.001 and.003, respectively), and granulocyte microparticle numbers correlated with systolic blood pressure (P =.05).

Conclusion: Numbers of T-cell and granulocyte microparticles are increased in preeclampsia. Whether these altered microparticle numbers cause vascular dysfunction in preeclampsia or are a consequence of the disease remains to be established.

MeSH terms

  • Adult
  • Blood Platelets / pathology*
  • Female
  • Flow Cytometry
  • Granulocytes / pathology*
  • Humans
  • Pancreatic Elastase / blood
  • Pre-Eclampsia / blood*
  • Pre-Eclampsia / pathology
  • Pregnancy
  • T-Lymphocytes / pathology*

Substances

  • Pancreatic Elastase