High glutamate decreases S100B secretion stimulated by serum deprivation in astrocytes

Neuroreport. 2002 Aug 27;13(12):1533-5. doi: 10.1097/00001756-200208270-00009.

Abstract

S100B is a calcium-binding protein expressed and secreted by astrocytes, playing a neurotrophic role in neighboring cells. A protective role of the S100B against glutamate-induced excitotoxicity has recently been proposed. We investigated S100B secretion in rat hippocampal astrocytes exposed to high concentrations of glutamate during serum deprivation (stimulated condition) or not (basal condition), for 30 min. Glutamate at 1 mM had no effect on basal secretion of S100B, but it decreased S100B secretion in serum-deprived astrocytes after 1 h. Secretion was inhibited by Rp-cAMPS or H89. In addition, serum deprivation was accompanied by a transitory increase of intracellular content of cAMP. Our results suggest that high levels of glutamate in a serum-deprived condition could impair S100B secretion from hippocampal astrocytes.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Astrocytes / cytology
  • Astrocytes / drug effects
  • Astrocytes / metabolism*
  • Calcium-Binding Proteins / metabolism*
  • Cells, Cultured
  • Culture Media, Serum-Free / pharmacology
  • Cyclic AMP / metabolism
  • Cycloheximide / pharmacology
  • Glutamic Acid / pharmacology*
  • Nerve Growth Factors / metabolism*
  • Neurotoxins / pharmacology
  • Protein Synthesis Inhibitors / pharmacology
  • Rats
  • Rats, Wistar
  • S100 Calcium Binding Protein beta Subunit
  • S100 Proteins*

Substances

  • Calcium-Binding Proteins
  • Culture Media, Serum-Free
  • Nerve Growth Factors
  • Neurotoxins
  • Protein Synthesis Inhibitors
  • S100 Calcium Binding Protein beta Subunit
  • S100 Proteins
  • S100b protein, rat
  • Glutamic Acid
  • Cycloheximide
  • Cyclic AMP