Interleukin 15 controls both proliferation and survival of a subset of memory-phenotype CD8(+) T cells

J Exp Med. 2002 Oct 7;196(7):935-46. doi: 10.1084/jem.20020772.

Abstract

Previous work has shown that memory-phenotype CD44(hi) CD8(+) cells are controlled by a cytokine, interleukin (IL)-15. However, the dependency of CD44(hi) CD8(+) cells on IL-15 is partial rather than complete. Here, evidence is presented that CD44(hi) CD8(+) cells comprise a mixed population of IL-15-dependent and IL-15-independent cells. The major subset of CD122(hi) CD44(hi) CD8(+) cells is heavily dependent on IL-15 by three different parameters, namely (1) "bystander" proliferation induced via IFN-induced stimulation of the innate immune system, (2) normal "background" proliferation, and (3) T cell survival; IL-15 dependency is most extreme for the Ly49(+) subset of CD122(hi) CD44(hi) CD8(+) cells. In contrast to CD122(hi) cells, the CD122(lo) subset of CD44(hi) CD8(+) cells is IL-15 independent; likewise, being CD122(lo), CD44(hi) CD4(+) cells are IL-15 independent. Thus, subsets of memory-phenotype T cells differ radically in their sensitivity to IL-15.

Publication types

  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Adoptive Transfer
  • Animals
  • Cell Division / drug effects
  • Cell Division / immunology*
  • Cell Separation / methods
  • Cell Survival / drug effects
  • Cell Survival / immunology*
  • Flow Cytometry
  • Hyaluronan Receptors / analysis
  • Immunologic Memory
  • Interleukin-15 / pharmacology*
  • Interleukin-2 / deficiency
  • Interleukin-2 / genetics
  • Interleukin-2 / immunology
  • Lymphocyte Activation
  • Mice
  • Mice, Inbred C57BL
  • Mice, Inbred Strains
  • Mice, Knockout
  • T-Lymphocyte Subsets / cytology
  • T-Lymphocyte Subsets / drug effects
  • T-Lymphocyte Subsets / immunology
  • T-Lymphocytes / cytology
  • T-Lymphocytes / drug effects
  • T-Lymphocytes / immunology*

Substances

  • Hyaluronan Receptors
  • Interleukin-15
  • Interleukin-2