Association of GPI-anchored protein TAG-1 with src-family kinase Lyn in lipid rafts of cerebellar granule cells

Neurochem Res. 2002 Aug;27(7-8):823-9. doi: 10.1023/a:1020265225916.

Abstract

We have demonstrated that antibody-mediated crosslinking of GPI-anchored TAG-1 induced activation of src-family kinase Lyn and rapid tyrosine phosphorylation of an 80-kDa protein (p80), a putative substrate for Lyn, in the lipid raft fraction prepared from primary cerebellar cultures, suggesting the functional association of TAG-1 with Lyn in lipid rafts of the rat cerebellum. In this study, the association was confirmed using a cDNA expression system. TAG-1-expressing CHO transfectants exhibited enhanced self-aggregation and promoted neurite outgrowth of primary cerebellar cultures as a culture substrate. The anti-TAG-1 antibody co-immunoprecipitated Lyn with TAG-1 and induced co-patching of TAG-1 with Lyn in both TAG-1 and Lyn-expressing CHO transfectants. Density gradient analysis revealed that TAG-1 is present in the lipid raft fraction of the CHO transfectants. Furthermore, pretreatment with a sphingolipid biosynthesis inhibitor ISP-1 reduced the extent of tyrosine phosphorylation of p80 by the antibody-mediated crosslinking of TAG-1. Immunocytochemical study showed that both TAG-1 and Lyn are present in cerebellar granule cells. These observations suggest that TAG-1 associates with Lyn in lipid rafts of rat cerebellar granule cells.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • CHO Cells
  • Cell Adhesion Molecules, Neuronal / genetics
  • Cell Adhesion Molecules, Neuronal / metabolism*
  • Cells, Cultured
  • Cerebellum / cytology
  • Cerebellum / enzymology
  • Cerebellum / metabolism*
  • Contactin 2
  • Cricetinae
  • Cytoplasmic Granules / enzymology
  • Cytoplasmic Granules / metabolism*
  • DNA, Complementary
  • Glycosylphosphatidylinositols / metabolism*
  • Lipid Metabolism*
  • Precipitin Tests
  • Sialyltransferases / genetics
  • Transfection
  • src-Family Kinases / metabolism*

Substances

  • Cell Adhesion Molecules, Neuronal
  • Cntn2 protein, rat
  • Contactin 2
  • DNA, Complementary
  • Glycosylphosphatidylinositols
  • Sialyltransferases
  • alpha-N-acetylneuraminate alpha-2,8-sialyltransferase
  • lyn protein-tyrosine kinase
  • src-Family Kinases