Inhibition of intracellular proliferation of Leishmania parasites in vitro and suppression of skin lesion development in BALB/c mice by a novel lipid A analog (ONO-4007)

Am J Trop Med Hyg. 2002 Aug;67(2):184-90. doi: 10.4269/ajtmh.2002.67.184.

Abstract

A synthetic lipid A analog (ONO-4007) exhibits antileishmanial activity by activating Leishmania-infected macrophages in experimental leishmaniasis. In the present in vitro study, ONO-4007 at concentrations between 0.01 and 1.00 mg/mL markedly inhibited the proliferation of Leishmania major and L. amazonensis promastigotes. Ultrastructurally, L. major-infected macrophages showed degenerated intracellular amastigotes after exposure to ONO-4007. Leishmania-infected macrophages treated with ONO-4007 showed poorly developed parasitophorous vacuoles. High levels of tumor necrosis factor-alpha were induced by ONO-4007 in Leishmania-infected macrophages. In this in vivo study, L. amazonensis-infected BALB/c mice were treated with a dose of 30 mg/kg of ONO-4007 by perilesional and peritoneal injections. The skin lesion size was assessed before treatment with ONO-4007 and at eight weeks after injection. The lesion size was significantly suppressed in mice perilesionally injected with ONO-4007 (P < 0.01) compared with the controls. The data from our present in vitro and in vivo studies indicate that ONO-4007 has an antileishmanial effect.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Antiprotozoal Agents / chemistry
  • Antiprotozoal Agents / pharmacology*
  • Antiprotozoal Agents / therapeutic use*
  • Cell Line
  • Enzyme-Linked Immunosorbent Assay
  • Female
  • Gene Expression Regulation / drug effects
  • Leishmania / drug effects*
  • Leishmania / growth & development*
  • Leishmania / ultrastructure
  • Leishmania major / drug effects
  • Leishmania major / growth & development
  • Leishmania major / ultrastructure
  • Leishmaniasis / drug therapy*
  • Leishmaniasis / parasitology
  • Lipid A / analogs & derivatives*
  • Lipid A / chemistry
  • Lipid A / pharmacology*
  • Lipid A / therapeutic use*
  • Macrophages / drug effects
  • Macrophages / parasitology
  • Macrophages / ultrastructure
  • Mice
  • Mice, Inbred BALB C
  • Specific Pathogen-Free Organisms
  • Time Factors
  • Tumor Necrosis Factor-alpha / biosynthesis

Substances

  • Antiprotozoal Agents
  • Lipid A
  • ONO 4007
  • Tumor Necrosis Factor-alpha