Revealing lymphoma growth and the efficacy of immune cell therapies using in vivo bioluminescence imaging

Blood. 2003 Jan 15;101(2):640-8. doi: 10.1182/blood-2002-06-1751. Epub 2002 Sep 26.

Abstract

Cancer therapeutics have achieved success in the treatment of a variety of malignancies, however, relapse of disease from small numbers of persistent tumor cells remains a major obstacle. Advancement of treatment regimens that effectively control minimal residual disease and prevent relapse would be greatly accelerated if sensitive and noninvasive assays were used to quantitatively assess tumor burden in animal models of minimal residual disease that are predictive of the human response. In vivo bioluminescence imaging (BLI) is an assay for the detection of small numbers of cells noninvasively and enables the quantification of tumor growth within internal organs. Fusion genes that encode bioluminescent and fluorescent reporter proteins effectively couple the powerful in vivo capabilities of BLI with the subset-discriminating capabilities of fluorescence-activated cell sorting. We labeled 2 murine lymphoma cell lines with dual function reporter genes and monitored radiation and chemotherapy as well as immune-based strategies that employ the tumorcidal activity of ex vivo-expanded CD8(+) natural killer (NK)-T cells. Using BLI we were able to visualize the entire course of malignant disease including engraftment, expansion, metastasis, response to therapy, and unique patterns of relapse. We also labeled the effector NK-T cells and monitored their homing to the sites of tumor growth followed by tumor eradication. These studies reveal the efficacy of immune cell therapies and the tempo of NK-T cell trafficking in vivo. The complex cellular processes in bone marrow transplantation and antitumor immunotherapy, previously inaccessible to investigation, can now be revealed in real time in living animals.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Animals
  • Antineoplastic Agents / pharmacology
  • Cell Division / drug effects
  • Cell Division / genetics
  • Cell Division / radiation effects
  • Diagnostic Imaging / methods*
  • Female
  • Genes, Reporter
  • Immunotherapy, Adoptive / methods*
  • Killer Cells, Lymphokine-Activated / metabolism
  • Killer Cells, Lymphokine-Activated / transplantation
  • Luminescent Measurements
  • Lymphoma / diagnosis
  • Lymphoma / pathology*
  • Lymphoma / therapy*
  • Mice
  • Mice, Inbred BALB C
  • Mice, Inbred C57BL
  • Neoplasm Metastasis / diagnosis
  • Neoplasm Metastasis / pathology
  • Radiotherapy
  • Transduction, Genetic

Substances

  • Antineoplastic Agents