The destruction box of the cyclin Clb2 binds the anaphase-promoting complex/cyclosome subunit Cdc23

Arch Biochem Biophys. 2002 Nov 15;407(2):189-95. doi: 10.1016/s0003-9861(02)00467-8.

Abstract

Properly regulated cyclin proteolysis is critical for normal cell cycle progression. A nine-amino acid peptide motif called the destruction box (D box) is present at the N terminus of the yeast mitotic cyclins. This short sequence is required for cyclin ubiquitination and subsequent proteolysis. The anaphase-promoting complex/cyclosome (APC/C) is a multisubunit E3 required for cyclin ubiquitination. We have tested the D box of five mitotic cyclins for interaction with six APC/C subunits. The APC/C subunit Cdc23, but not five other subunits tested, interacted by two-hybrid analysis with the N terminus of wild-type Clb2. None of these subunits interacted with the N termini of the cyclins Clb1, Clb3, or Clb5. Mutations in the D box sequences of Clb2 inhibited interaction with Cdc23 both in vivo and in vitro. Our results provide the first evidence for a direct interaction between an APC/C substrate (Clb2) and an APC/C subunit (Cdc23).

MeSH terms

  • Amino Acid Motifs
  • Anaphase-Promoting Complex-Cyclosome
  • Apc8 Subunit, Anaphase-Promoting Complex-Cyclosome
  • Binding Sites
  • Cell Cycle Proteins / metabolism*
  • Cyclin B / chemistry*
  • Cyclin B / genetics
  • Cyclin B / metabolism*
  • Fungal Proteins / chemistry
  • Fungal Proteins / genetics
  • Fungal Proteins / metabolism
  • Ligases / chemistry
  • Ligases / metabolism*
  • Mutation
  • Protein Binding
  • Protein Subunits
  • Saccharomyces cerevisiae Proteins*
  • Two-Hybrid System Techniques
  • Ubiquitin-Protein Ligase Complexes*

Substances

  • Apc8 Subunit, Anaphase-Promoting Complex-Cyclosome
  • CDC23 protein, S cerevisiae
  • CLB2 protein, S cerevisiae
  • Cell Cycle Proteins
  • Cyclin B
  • Fungal Proteins
  • Protein Subunits
  • Saccharomyces cerevisiae Proteins
  • Ubiquitin-Protein Ligase Complexes
  • Anaphase-Promoting Complex-Cyclosome
  • Ligases