Practical importance of neuroprotection in Parkinson's disease

J Neurol. 2002 Oct:249 Suppl 3:III/53-6. doi: 10.1007/s00415-002-1311-2.

Abstract

Consensus could be reached that there is overwhelming evidence of preclinical neuroprotection. However, the evidence of neuroprotection/neurorescue under clinical conditions is limited. Lessons from clinical trials designed to show neuroprotection (selegiline, amantadine, dopamine agonists) demonstrate that with the drugs available neuroprotection/neurorescue has to start as early as possible. A PET-controlled clinical trial with ropinirole shows that there seems to be a good chance for neuroprotection in the early phase of Parkinson's disease in patients treated from the very beginning of the disease while there is no such benefit in patients with a late start of a neuroprotective therapeutic strategy. Also long-term clinical neuroprotection cannot be reached. Complicating factors to demonstrate clinical neuroprotection are discussed.

Publication types

  • Review

MeSH terms

  • Amantadine / therapeutic use
  • Antiparkinson Agents / therapeutic use*
  • Dopamine Agonists / therapeutic use
  • Humans
  • Indoles / therapeutic use
  • Neuroprotective Agents / therapeutic use*
  • Parkinson Disease / therapy*
  • Selegiline / therapeutic use

Substances

  • Antiparkinson Agents
  • Dopamine Agonists
  • Indoles
  • Neuroprotective Agents
  • ropinirole
  • Selegiline
  • Amantadine