p53RDL1 regulates p53-dependent apoptosis

Nat Cell Biol. 2003 Mar;5(3):216-23. doi: 10.1038/ncb943.

Abstract

Although a number of targets for p53 have been reported, the mechanism of p53-dependent apoptosis still remains to be elucidated. Here we report a new p53 target-gene, designated p53RDL1 (p53-regulated receptor for death and life; also termed UNC5B). The p53RDL1 gene product contains a cytoplasmic carboxy-terminal death domain that is highly homologous to rat Unc5H2, a dependence receptor involved in the regulation of apoptosis, as well as in axon guidance and migration of neural cells. We found that p53RDL1 mediated p53-dependent apoptosis. Conversely, when p53RDL1 interacted with its ligand, Netrin-1, p53-dependent apoptosis was blocked. Therefore, p53RDL1 seems to be a previously un-recognized target of p53 that may define a new pathway for p53-dependent apoptosis. We suggest that p53 might regulate the survival of damaged cells by balancing the regulation of Netrin-p53RDL1 signalling, and cell death through cleavage of p53RDL1 for apoptosis.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Apoptosis / physiology*
  • Base Sequence
  • Cloning, Molecular
  • DNA Primers
  • DNA, Complementary
  • Humans
  • Immunohistochemistry
  • Molecular Sequence Data
  • Nerve Growth Factors / physiology
  • Netrin Receptors
  • Netrin-1
  • Rats
  • Receptors, Cell Surface / genetics
  • Receptors, Cell Surface / physiology*
  • Signal Transduction
  • Tumor Cells, Cultured
  • Tumor Suppressor Protein p53 / physiology*
  • Tumor Suppressor Proteins

Substances

  • DNA Primers
  • DNA, Complementary
  • NTN1 protein, human
  • Nerve Growth Factors
  • Netrin Receptors
  • Ntn1 protein, rat
  • Receptors, Cell Surface
  • Tumor Suppressor Protein p53
  • Tumor Suppressor Proteins
  • Netrin-1

Associated data

  • GENBANK/AB096256
  • GENBANK/AC006187
  • GENBANK/AL359384
  • GENBANK/AL359832