IL-15R alpha expression on CD8+ T cells is dispensable for T cell memory

Proc Natl Acad Sci U S A. 2003 Apr 15;100(8):4724-9. doi: 10.1073/pnas.0737048100. Epub 2003 Apr 1.

Abstract

The generation and maintenance of immunological memory requires the activation, expansion, and persistent proliferation of antigen-specific T cells. Recent work suggests that IL-15 may be important for this process. Surprisingly, we now find that expression of the high-affinity receptor for IL-15, IL-15R alpha, on T cells is dispensable for the generation or maintenance of memory CD8(+) T cells. By contrast, IL-15R alpha expression on cells other than T cells is absolutely critical for this function. These findings may be related to IL-15R alpha's ability to present IL-15 in trans to low-affinity IL-15R beta gamma(c) receptors on memory CD8(+) T cells. These unexpected results provide insights into how IL-15R alpha supports memory CD8(+) T cells.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Adjuvants, Immunologic / pharmacology
  • Adoptive Transfer
  • Animals
  • CD8-Positive T-Lymphocytes / cytology
  • CD8-Positive T-Lymphocytes / drug effects
  • CD8-Positive T-Lymphocytes / immunology*
  • Cell Division
  • Immunologic Memory*
  • Mice
  • Mice, Congenic
  • Mice, Inbred C57BL
  • Mice, Knockout
  • Poly I-C / pharmacology
  • Radiation Chimera
  • Radiation Tolerance / immunology
  • Receptors, Interleukin-15
  • Receptors, Interleukin-2 / deficiency
  • Receptors, Interleukin-2 / genetics
  • Receptors, Interleukin-2 / metabolism*
  • Signal Transduction

Substances

  • Adjuvants, Immunologic
  • Il15ra protein, mouse
  • Receptors, Interleukin-15
  • Receptors, Interleukin-2
  • Poly I-C