[An experimental study on the role of indirect antigen presentation pathway on the immunogenetic of epidermal cells]

Zhonghua Shao Shang Za Zhi. 2003 Feb;19(1):15-7.
[Article in Chinese]

Abstract

Objective: To explore the role of indirect antigen presentation pathway on the immunogenecity of epidermal cells.

Methods: Human epidermal cells (HEC), allogeneic human peripheral blood lymphocytes (PBL) and mononuclear cells (PBM, including monocytes) were isolated and cultured in vitro. HECs were transfected by human-originated CTLA4Ig-adenovirus vector. The CTLA4Ig expression was observed. Allogeneic PBLs or PBMs were added to the transfected and non-transfected HECs with simple cultured PBLs and PBMs as the control. The proliferation of PBL and PBM was determined by (3)H-TdR incooperation.

Results: HECs could be successfully transfected by CTLA4Ig-adenovirus vector and expressed corresponding proteins. The non-transfected HECs could stimulate slight proliferation of allogeneic PBLs (P < 0.05) and stimulate remarkable proliferation of PBMs (including monocytes) (P < 0.05). The proliferation reaction of PBLs and PBMs decreased significantly (P < 0.05) after being stimulated by HEC which was modulated by CTLA4Ig genes.

Conclusion: Indirect antigen presentation pathway might play important roles in the HEC immunogenicity which could be evidently inhibited by CTLA4Ig.

MeSH terms

  • Adenoviridae / genetics
  • Antigen Presentation / immunology*
  • Antigen Presentation / physiology
  • Antigens, CD
  • Antigens, Differentiation / genetics
  • Antigens, Differentiation / immunology
  • CTLA-4 Antigen
  • Cell Division / immunology
  • Cells, Cultured
  • Coculture Techniques
  • Epidermal Cells
  • Epidermis / immunology*
  • Epidermis / metabolism
  • Genetic Vectors / genetics
  • Humans
  • Leukocytes, Mononuclear / cytology
  • Leukocytes, Mononuclear / immunology
  • Lymphocytes / cytology
  • Lymphocytes / immunology
  • Signal Transduction
  • Transfection

Substances

  • Antigens, CD
  • Antigens, Differentiation
  • CTLA-4 Antigen
  • CTLA4 protein, human