Effects of ethanol on anti-saccade task performance

Exp Brain Res. 2003 May;150(1):68-74. doi: 10.1007/s00221-003-1400-1. Epub 2003 Mar 4.

Abstract

It has been shown that saccade-related neurons in the superior colliculus (SC) display an increased level of prestimulus activity and a higher stimulus-related burst in action potentials preceding direction errors in the anti-saccade task compared with correct anti-saccades. From this, it has been hypothesized that errors occur when the incoming visual signal in the SC passes a threshold and triggers a reflexive saccade. This hypothesis predicts that an attenuated visual signal will reduce the number of direction errors. Since ethanol has been shown to have a suppressive effect on cortical visual event-related potentials (ERPs), the purpose of the present study was to investigate the effects of moderate ethanol consumption on anti-saccade performance. Under both placebo and ethanol conditions, we recorded ERPs and measured eye movements in male subjects during the performance of an anti-saccade task in which the fixation point disappeared 200 ms prior to stimulus presentation. Compared with the placebo condition, we found in the ethanol condition: (1). a decrease in ERP amplitudes during the gap period and after stimulus presentation, (2). an increase in the latencies of anti-saccades, and (3). a decrease in the percentage of direction errors. These data demonstrate the effects of ethanol on anti-saccade task performance and provide further support for the hypothesis that errors in the anti-saccade task are triggered by the incoming visual signal.

Publication types

  • Clinical Trial
  • Controlled Clinical Trial
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adult
  • Ethanol / pharmacology*
  • Evoked Potentials / drug effects*
  • Evoked Potentials / physiology
  • Fixation, Ocular / drug effects
  • Fixation, Ocular / physiology
  • Humans
  • Male
  • Models, Neurological
  • Neural Inhibition / drug effects
  • Neural Inhibition / physiology
  • Neurons / drug effects*
  • Neurons / physiology
  • Ocular Motility Disorders / chemically induced
  • Ocular Motility Disorders / physiopathology
  • Psychomotor Performance / drug effects*
  • Psychomotor Performance / physiology
  • Reaction Time / drug effects
  • Reaction Time / physiology
  • Saccades / physiology*
  • Superior Colliculi / drug effects*
  • Superior Colliculi / physiology
  • Visual Pathways / drug effects*
  • Visual Pathways / physiology

Substances

  • Ethanol