Helicobacter pylori stimulates inducible nitric oxide synthase in diverse topographical patterns in various gastroduodenal disorders

Dig Dis Sci. 2003 Apr;48(4):636-43. doi: 10.1023/a:1022855818944.

Abstract

Overproduction of nitric oxide by inducible nitric oxide synthase (iNOS) acts cytotoxically and contributes to inflammation. We explored the roles of iNOS in the pathogenesis of Helicobacter pylori-associated diseases. Using reverse-transcribed PCR, we examined topographical patterns of iNOS mRNA expression in the gastroduodenal mucosa in H. pylori-negative controls and H. pylori-positive patients with duodenal ulcer (DU), gastric ulcer (GU), and ulcer-free gastritis. iNOS expression showed topographical variations among the tested disorders. As compared to controls, DU had a significantly higher expression of iNOS mRNA in the duodenum, GU in the antrum and duodenum, and gastritis in the antrum and corpus. H. pylori eradication yielded a significant reduction of iNOS mRNA in the duodenum of DU and in the antrum of GU. Diverse topographical patterns of H. pylori-induced iNOS expression may contribute to mechanisms by which H. pylori elicits different clinical disorders.

Publication types

  • Clinical Trial
  • Randomized Controlled Trial

MeSH terms

  • Amoxicillin / administration & dosage
  • Anti-Ulcer Agents / administration & dosage
  • Biopsy
  • Clarithromycin / administration & dosage
  • Drug Therapy, Combination
  • Duodenal Ulcer / drug therapy
  • Duodenal Ulcer / enzymology
  • Duodenal Ulcer / microbiology*
  • Endoscopy, Digestive System
  • Enzyme Induction / drug effects
  • Enzyme Induction / physiology
  • Gastric Mucosa / enzymology
  • Gastric Mucosa / microbiology
  • Gastric Mucosa / pathology
  • Gastritis / drug therapy
  • Gastritis / enzymology
  • Gastritis / microbiology*
  • Gene Expression Regulation, Enzymologic / drug effects
  • Gene Expression Regulation, Enzymologic / physiology
  • Helicobacter pylori / drug effects
  • Helicobacter pylori / physiology*
  • Humans
  • Immunoenzyme Techniques
  • Intestinal Mucosa / enzymology
  • Intestinal Mucosa / microbiology
  • Intestinal Mucosa / pathology
  • Nitric Oxide Synthase / biosynthesis*
  • Nitric Oxide Synthase / genetics
  • Nitric Oxide Synthase Type II
  • Omeprazole / administration & dosage
  • Pyloric Antrum / enzymology
  • Pyloric Antrum / microbiology
  • Pyloric Antrum / pathology
  • RNA, Messenger / genetics

Substances

  • Anti-Ulcer Agents
  • RNA, Messenger
  • Amoxicillin
  • NOS2 protein, human
  • Nitric Oxide Synthase
  • Nitric Oxide Synthase Type II
  • Clarithromycin
  • Omeprazole