Increase in IP3 and intracellular Ca2+ induced by phosphate depletion in LLC-PK 1 cells

Biochem Biophys Res Commun. 2003 May 30;305(2):287-91. doi: 10.1016/s0006-291x(03)00750-2.

Abstract

The mechanisms by which Pi depletion rapidly regulates gene expression and cellular function have not been clarified. Here, we found a rapid increase in intracellular ionized calcium [Ca(2+)](i) by phosphate depletion in LLC-PK(1) cells using confocal microscopy with the green-fluorescence protein based calcium indicator "yellow cameleon 2.1." The increase of [Ca(2+)](i) was observed in the presence or absence of extracellular Ca(2+). At the same time, an approximately twofold increase in intracellular inositol 1,4,5-triphosphate (IP(3)) occurred in response to the acute Pi depletion in the medium. Furthermore, 2-aminoethoxydiphenyl borate completely blocked the [Ca(2+)](i) increase induced by Pi depletion. These results suggest that Pi depletion causes IP(3)-mediated release of Ca(2+) from intracellular Ca(2+) pools and rapidly increases [Ca(2+)](i) in LLC-PK(1) cells.

Publication types

  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Animals
  • Boron Compounds / pharmacology
  • Calcium / metabolism*
  • Cell Line
  • Cytoplasm / metabolism
  • Inositol 1,4,5-Trisphosphate / metabolism*
  • Kidney Tubules, Proximal / drug effects
  • Kidney Tubules, Proximal / metabolism*
  • Kinetics
  • Microscopy, Confocal
  • Phosphates / physiology*
  • Rats

Substances

  • Boron Compounds
  • Phosphates
  • Inositol 1,4,5-Trisphosphate
  • 2-aminoethoxydiphenyl borate
  • Calcium