Abstract
The Toll-like receptors (TLRs) are recently discovered germline-encoded receptors on APCs that are critically important in innate immune recognition of microbial pathogens. However, their role in solid-organ transplantation is unknown. To explore this role, we employed a skin allograft model using mice with targeted deletion of the universal TLR signal adaptor protein, MyD88. We report that minor antigen-mismatched (HY-mismatched) allograft rejection cannot occur in the absence of MyD88 signaling. Furthermore, we show that the inability to reject these allografts results from a reduced number of mature DCs in draining lymph nodes, leading to impaired generation of anti-graft-reactive T cells and impaired Th1 immunity. Hence, this work demonstrates that TLRs can be activated in a transplant setting and not solely by infections. These results link innate immunity to the initiation of the adaptive alloimmune response.
Publication types
-
Research Support, U.S. Gov't, P.H.S.
MeSH terms
-
Acute Disease
-
Adaptor Proteins, Signal Transducing
-
Animals
-
Antigens, CD / immunology
-
Antigens, CD / metabolism
-
Antigens, Differentiation / genetics
-
Antigens, Differentiation / immunology
-
Antigens, Differentiation / physiology*
-
Cell Count
-
Dendritic Cells / cytology
-
Dendritic Cells / immunology
-
Drosophila Proteins*
-
Female
-
Graft Rejection / genetics
-
Graft Rejection / immunology*
-
H-Y Antigen / immunology
-
Homozygote
-
Immunity, Innate / genetics
-
Immunity, Innate / immunology
-
Interferon-gamma / metabolism
-
Interleukin-4 / metabolism
-
Male
-
Membrane Glycoproteins / metabolism*
-
Mice
-
Mice, Mutant Strains
-
Myeloid Differentiation Factor 88
-
Receptors, Cell Surface / metabolism*
-
Receptors, Immunologic / genetics
-
Receptors, Immunologic / immunology
-
Receptors, Immunologic / physiology*
-
Skin Transplantation / immunology
-
T-Lymphocytes / immunology
-
Th1 Cells / immunology
-
Th2 Cells / immunology
-
Toll-Like Receptors
-
Transplantation Tolerance / immunology*
Substances
-
Adaptor Proteins, Signal Transducing
-
Antigens, CD
-
Antigens, Differentiation
-
Drosophila Proteins
-
H-Y Antigen
-
Membrane Glycoproteins
-
Myd88 protein, mouse
-
Myeloid Differentiation Factor 88
-
Receptors, Cell Surface
-
Receptors, Immunologic
-
Toll-Like Receptors
-
Interleukin-4
-
Interferon-gamma