[Inhibition of HBV DNA replication and expression in 2.2.15 hepatoma cells infected with AFP-mediated HBX antisense RNA]

Zhonghua Gan Zang Bing Za Zhi. 2003 May;11(5):291-4.
[Article in Chinese]

Abstract

Objective: To study the specific expression of the antisense RNA against hepatitis B virus X (HBX) gene in hepatoblastoma cell line and its anti -HBV activity.

Methods: HBX gene (nt.1370-1827) was amplified by PCR, then cloned into EB virus vector pEBAF which contained human alpha-fetoprotein promoter and enhancer. After transfected into 2.2.15 hepatoma cells and ECV304 human endothelial cells by lipofectin, northern blot, ELISA and real-time qualitative PCR were carried out to assay the expression of HBX mRNA, HBV antigens and HBV DNA level, respectively.

Results: The HBX antisense RNA expression vector pEBAF-as-HBX which could be expressed specifically in 2.2.15 hepatoblastoma cells was successfully constructed. Both HBV DNA level and the expressions of hepatitis B virus surface antigen (HBsAg) and e antigen (HBeAg) in 2.2.15 hepatoblastoma cells were inhibited by pEBAF-as-HBX. Compared with those in sense control (pEBAF-s-HBX), the inhibitory rates of HBsAg, HBeAg, and HBV DNA were 37.9%, 36.8%, and 25%, respectively.

Conclusions: The pEBAF-as-HBX expression vector may lead to targeted-expression of HBX antisense RNA in hepatoma cells and shows great inhibition effect on HBV.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Carcinoma, Hepatocellular / genetics
  • Carcinoma, Hepatocellular / pathology
  • Carcinoma, Hepatocellular / virology*
  • Cell Line, Tumor
  • DNA Replication
  • Enhancer Elements, Genetic / genetics
  • Gene Expression Regulation, Viral / drug effects
  • Genetic Therapy / methods
  • Hepatitis B virus / genetics*
  • Hepatitis B virus / physiology
  • Humans
  • Liver Neoplasms / genetics
  • Liver Neoplasms / pathology
  • Liver Neoplasms / virology*
  • Promoter Regions, Genetic / genetics
  • RNA, Antisense / pharmacology*
  • Trans-Activators / biosynthesis*
  • Trans-Activators / genetics
  • Transcriptional Activation
  • Transfection
  • Viral Regulatory and Accessory Proteins
  • alpha-Fetoproteins / genetics*

Substances

  • RNA, Antisense
  • Trans-Activators
  • Viral Regulatory and Accessory Proteins
  • alpha-Fetoproteins
  • hepatitis B virus X protein