B cell apoptosis accelerates the onset of murine lupus

Eur J Immunol. 2003 Jun;33(6):1603-12. doi: 10.1002/eji.200323665.

Abstract

To investigate whether the increased rate of lymphocyte apoptosis in systemic lupus erythematosus is involved in the onset of the disease, apoptotic or necrotic T or B lymphocytes from various cell lines were injected intraperitoneally into pre-autoimmune (NZBxNZW)F1 mice (BW) and non-autoimmune BALB/c mice. The intraperitoneal production of cytokines and chemokines, the specific T cell response in the spleen, and the production of anti-histone and anti-dsDNA Ab were investigated. The onset of the disease was characterized by creatinine levels and evaluation of glomerular IgG deposits. In BW, but not in BALB/c mice, injection of apoptotic and not necrotic cells up-regulated IL-6 and IL-10 in resident macrophages. Administration of apoptotic cells augmented the number of Th2 and B lymphocytes recruited in the peritoneal cavity. Only the treatment with apoptotic B cells promoted a systemic Th2 autoimmune response to H2 histones, associated with earlier occurrence of high levels of anti-dsDNA autoantibodies, higher creatinine levels and more numerous glomerular IgG deposits than in BW controls not injected with apoptotic B cells. In genetically susceptible mice exposure to apoptotic of B, but not T, lymphocytes can elicit a Th2 response to H2 histones that helps B cell production of anti-dsDNA Ab and finally triggers the onset of lupus.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Antibodies, Antinuclear / analysis
  • Antibodies, Antinuclear / immunology
  • Apoptosis*
  • Autoantibodies / analysis
  • Autoantibodies / immunology
  • Autoantigens / immunology*
  • Autoimmune Diseases / blood
  • Autoimmune Diseases / immunology*
  • Autoimmune Diseases / pathology
  • B-Lymphocytes / pathology*
  • Chemokines / biosynthesis
  • Creatinine / blood
  • DNA / immunology
  • Disease Models, Animal
  • Disease Progression
  • Epitopes, T-Lymphocyte / immunology
  • Female
  • Histones / classification
  • Histones / immunology*
  • Immunization
  • Immunoglobulin G / analysis
  • Immunoglobulin G / immunology
  • Interferon-gamma / biosynthesis
  • Interferon-gamma / blood
  • Interleukin-4 / biosynthesis
  • Interleukin-4 / blood
  • Kidney Glomerulus / chemistry
  • Kidney Glomerulus / immunology
  • Kidney Glomerulus / pathology
  • Leukemia, T-Cell / immunology
  • Leukemia, T-Cell / pathology
  • Lupus Erythematosus, Systemic / blood
  • Lupus Erythematosus, Systemic / immunology*
  • Lupus Erythematosus, Systemic / pathology
  • Lupus Nephritis / blood
  • Lupus Nephritis / immunology
  • Lupus Nephritis / pathology
  • Lymphocyte Cooperation
  • Mice
  • Mice, Inbred BALB C
  • Mice, Inbred NZB
  • Phagocytosis
  • Plasmacytoma / immunology
  • Plasmacytoma / pathology
  • Spleen / immunology
  • T-Lymphocyte Subsets / immunology
  • Th2 Cells / immunology
  • Tumor Cells, Cultured

Substances

  • Antibodies, Antinuclear
  • Autoantibodies
  • Autoantigens
  • Chemokines
  • Epitopes, T-Lymphocyte
  • Histones
  • Immunoglobulin G
  • Interleukin-4
  • Interferon-gamma
  • DNA
  • Creatinine