Regulation of FcepsilonRI-mediated signaling by an adaptor protein STAP-2/BSK in rat basophilic leukemia RBL-2H3 cells

Biochem Biophys Res Commun. 2003 Jul 4;306(3):767-73. doi: 10.1016/s0006-291x(03)01042-8.

Abstract

Crosslinking of multivalent antigen bound IgE transduces FcepsilonRI mediated signaling cascades, which activate nonreceptor-type protein-tyrosine kinases and subsequent tyrosine phosphorylation of cellular proteins, and these are critical elements for degranulation in mast cells. We cloned a novel adaptor molecule, signal transducing adaptor protein (STAP)-2 containing PH and SH2-like domains as a c-fms interacting protein. STAP-2 was identical to a recently cloned adaptor molecule, BKS, a substrate of BRK (breast tumor kinase) tyrosine kinase, although its function is still unknown. To examine a novel function of STAP-2/BSK, we expressed STAP-2/BSK or its mutants in rat basophilic leukemia RBL-2H3 cells. Overexpression of STAP-2/BSK resulted in a suppression of FcepsilonRI-mediated calcium mobilization and degranulation. FcepsilonRI-induced tyrosine phosphorylation of phospholipase C-gamma (PLC-gamma) but not Syk was significantly suppressed in these cells. Furthermore, STAP-2/BSK associated with PLC-gamma in vivo. These data indicate that STAP-2/BSK negatively controls the FcepsilonRI-mediated calcium mobilization and degranulation by direct modulation of tyrosine phosphorylation of PLC-gamma.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adaptor Proteins, Signal Transducing*
  • Adaptor Proteins, Vesicular Transport / genetics
  • Adaptor Proteins, Vesicular Transport / metabolism*
  • Animals
  • Calcium / metabolism
  • DNA-Binding Proteins / genetics
  • DNA-Binding Proteins / metabolism
  • Enzyme Precursors / metabolism
  • Genes, Reporter
  • Humans
  • Intracellular Signaling Peptides and Proteins
  • Leukemia, Basophilic, Acute
  • Mast Cells / cytology
  • Mast Cells / metabolism
  • NFATC Transcription Factors
  • Nuclear Proteins*
  • Phospholipase C gamma
  • Phosphoproteins*
  • Protein-Tyrosine Kinases / metabolism
  • Rats
  • Receptors, IgE / genetics
  • Receptors, IgE / metabolism*
  • Recombinant Fusion Proteins / metabolism
  • Signal Transduction / immunology*
  • Signal Transduction / physiology
  • Syk Kinase
  • Transcription Factors / genetics
  • Transcription Factors / metabolism
  • Tumor Cells, Cultured
  • Type C Phospholipases / metabolism

Substances

  • Adaptor Proteins, Signal Transducing
  • Adaptor Proteins, Vesicular Transport
  • DNA-Binding Proteins
  • Enzyme Precursors
  • Intracellular Signaling Peptides and Proteins
  • NFATC Transcription Factors
  • Nuclear Proteins
  • Phosphoproteins
  • Receptors, IgE
  • Recombinant Fusion Proteins
  • STAP2 protein, human
  • Transcription Factors
  • Protein-Tyrosine Kinases
  • SYK protein, human
  • Syk Kinase
  • Syk protein, rat
  • Type C Phospholipases
  • Phospholipase C gamma
  • Calcium