T-588, a cognitive enhancer, stimulates in vivo phosphorylation of extracellular signal-regulated kinases in the hippocampus

Brain Res Cogn Brain Res. 2003 Jul;17(2):522-5. doi: 10.1016/s0926-6410(03)00142-3.

Abstract

Administration of (1R)-1-benzo[b]thiophen-5-yl-2-[2-(diethylamino)ethoxy]ethan-1-ol hydrochloride (T-588), a cognitive enhancer, like the acetylcholine esterase inhibitors physostigmine and tacrine, stimulated phosphorylation of extracellular signal-regulated kinases (ERK) in the mouse hippocampus. The effect of T-588 on ERK phosphorylation was persistent from 2 to 6 h after injection. Immunohistochemical study showed that T-588 stimulated neuronal ERK phosphorylation in rat hippocampal CA1 pyramidal subfield. These findings suggest that systemic T-588 stimulates the ERK kinase pathway in the hippocampal neurons.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Diethylamines / pharmacology*
  • Dose-Response Relationship, Drug
  • Hippocampus / drug effects*
  • Hippocampus / enzymology
  • Male
  • Mice
  • Mitogen-Activated Protein Kinases / metabolism*
  • Nootropic Agents / pharmacology*
  • Phosphorylation / drug effects
  • Rats
  • Rats, Wistar
  • Thiophenes / pharmacology*

Substances

  • Diethylamines
  • Nootropic Agents
  • Thiophenes
  • T 588
  • Mitogen-Activated Protein Kinases