Abstract
Conformational change in arrestin induced by receptor binding promotes its interaction with the majority of recently identified nonreceptor binding partners.
MeSH terms
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Arrestin / chemistry*
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Arrestin / metabolism*
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Binding Sites
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Enzyme Activation
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MAP Kinase Kinase Kinases / metabolism
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Mitogen-Activated Protein Kinase Kinases / metabolism
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Models, Biological
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Models, Molecular
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Protein Binding
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Protein Conformation
Substances
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Arrestin
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MAP Kinase Kinase Kinases
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Mitogen-Activated Protein Kinase Kinases