Inhibitory effect of human syncytiotrophoblast plasma membrane vesicles on Jurkat cells activated by phorbol ester and calcium ionophore

Cell Immunol. 1992 Jan;139(1):259-67. doi: 10.1016/0008-8749(92)90118-9.

Abstract

The effects of syncytiotrophoblast plasma membrane vesicles (STPM) on stimulated Jurkat leukemic T cells have been investigated. STPM inhibited IL-2 production and the expression of protein P55 of the IL-2 receptor (IL-2R P55), when Jurkat cells were stimulated by a combination of calcium ionophore A23187 (CaI) + phorbol 12-myristate 13-acetate (PMA). STPM also inhibited IL-2R P55 when cells were stimulated by PMA alone, a situation in which IL-2 production is negligible. On the other hand, STPM had no effect on the sustained mobilization of intracellular Ca2+ induced by CaI nor on the PKC-dependent CD3 down regulation induced by PMA. Finally STPM had no effect on intracellular cAMP levels. These results show that (i) the inhibitory effect of STPM on IL-2R P55 expression is independent of the inhibition of IL-2 production, and (ii) the inhibitory effects of STPM are at least partially independent of phosphatidylinositol 4,5-bisphosphate hydrolysis. They suggest that STPM affect a signaling pathway activated by PMA but possibly PKC independent.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Antigens, Differentiation, T-Lymphocyte / metabolism
  • CD3 Complex
  • Calcimycin / pharmacology
  • Calcium / physiology
  • Cell Membrane / immunology
  • Colforsin / pharmacology
  • Cyclic AMP / physiology
  • Cytoplasm / metabolism
  • Down-Regulation
  • Female
  • Humans
  • Interleukin-2 / biosynthesis
  • Lymphocyte Activation / drug effects
  • Pregnancy / immunology*
  • Protein Kinase C / metabolism
  • Receptors, Antigen, T-Cell / metabolism
  • Receptors, Interleukin-2 / metabolism
  • T-Lymphocytes / immunology*
  • Tetradecanoylphorbol Acetate / pharmacology
  • Trophoblasts / immunology*
  • Tumor Cells, Cultured

Substances

  • Antigens, Differentiation, T-Lymphocyte
  • CD3 Complex
  • Interleukin-2
  • Receptors, Antigen, T-Cell
  • Receptors, Interleukin-2
  • Colforsin
  • Calcimycin
  • Cyclic AMP
  • Protein Kinase C
  • Tetradecanoylphorbol Acetate
  • Calcium