Cholesterol-rich rabbit serum modulates beta-adrenergic receptor density of human lymphocytes. A possible role of LDL-cholesterol

Ann N Y Acad Sci. 1992 Apr 15:650:239-44. doi: 10.1111/j.1749-6632.1992.tb49130.x.

Abstract

The effect of in vitro treatment of human lymphocytes with rabbit cholesterol-rich serum (RCS) on the membrane microviscosity as well as on the beta-adrenergic receptor density has been investigated. RCS treatment of cells resulted in a 30% decrease of receptor density without any effect on membrane microviscosity. A complete recovery was observed incubating the RCS cells either with the "Active Lipids" (AL) or with heparin. The AL are a mixture of neutral lipids, phosphatidylcholine and phosphatidylethanolamine from hen egg yolk known to fluidify the cell membrane. The AL modified membrane microviscosity of control lymphocytes without altering their beta-receptor number. These observations support the proposition that beta-receptor density of human lymphocytes is not regulated by membrane microviscosity and suggest that probably low density lipoprotein-cholesterol complex is involved in such a regulation.

MeSH terms

  • Animals
  • Cell Membrane / metabolism
  • Cholesterol, LDL / metabolism*
  • Heparin / pharmacology
  • Humans
  • In Vitro Techniques
  • Lipids / pharmacology
  • Lymphocytes / metabolism*
  • Membrane Fluidity
  • Rabbits
  • Receptors, Adrenergic, beta / metabolism*

Substances

  • Cholesterol, LDL
  • Lipids
  • Receptors, Adrenergic, beta
  • Heparin