Proliferative activity in gastric cancer determined with cell cycle-related monoclonal antibodies Ki-67 and p105: analysis by flow cytometry

J Surg Oncol. 1992 Nov;51(3):174-8. doi: 10.1002/jso.2930510310.

Abstract

The proliferative activity of gastric cancer cells was determined by DNA flow cytometric (FCM) analysis and labeling rates of Ki-67 and monoclonal antibodies and proliferation-associated nuclear antigen (p105) autoantibodies in 28 patients with fresh human gastric cancer cells. By setting the cutoff line at the level as used in a negative control study without primary antibody in the same sample, the Ki-67 and p105 labeling rates were calculated by the dual fluorescence analysis. A total of 43 experiments was performed on FCM analysis for each antigen: 28 with Ki-67 and 15 with p105. The mean Ki-67 labeling rate of gastric cancer cells was 45.1% (13.9-76.3%). The Ki-67 labeling rates were significantly higher for larger size tumor, peritoneal metastasis, and advanced clinical stage. A significant correlation was found between Ki-67 labeling rate and p105 labeling rate (P < 0.05). Bivariate FCM may be an easy method for obtaining useful information of cell kinetics.

MeSH terms

  • Antibodies, Monoclonal
  • Antigens, Neoplasm / analysis*
  • Antigens, Neoplasm / immunology
  • Cell Cycle
  • DNA, Neoplasm / analysis
  • Flow Cytometry
  • Humans
  • Ki-67 Antigen
  • Neoplasm Proteins / analysis*
  • Neoplasm Proteins / immunology
  • Nuclear Proteins / analysis*
  • Nuclear Proteins / immunology
  • Proliferating Cell Nuclear Antigen
  • Stomach Neoplasms / chemistry
  • Stomach Neoplasms / pathology*

Substances

  • Antibodies, Monoclonal
  • Antigens, Neoplasm
  • DNA, Neoplasm
  • Ki-67 Antigen
  • Neoplasm Proteins
  • Nuclear Proteins
  • Proliferating Cell Nuclear Antigen