Ruthenium red affects the intrinsic fluorescence of the calcium-ATPase of skeletal sarcoplasmic reticulum

Biochim Biophys Acta. 1992 Jun 19;1100(3):321-8. doi: 10.1016/0167-4838(92)90488-y.

Abstract

We have studied the effect of Ruthenium red on the sarcoplasmic reticulum Ca(2+)-ATPase. Ruthenium red does not modify the Ca2+ pumping activity of the enzyme, despite its interaction with cationic binding sites on sarcoplasmic reticulum vesicles. Two pools of binding sites were distinguished. One pool (10 nmol/mg) is dependent upon the presence of micromolar Ca2+ and may therefore represent the high-affinity Ca2+ transport sites of the Ca(2+)-ATPase. However, Ruthenium red only slightly competes with Ca2+ on these sites. The other pool (15-17 nmol/mg) is characterized as low-affinity cation binding sites of sarcoplasmic reticulum, distinct from the Mg2+ site involved in the ATP binding to the Ca(2+)-ATPase. The interaction of Ruthenium red with these low-affinity cation binding sites, which may be located either on the Ca(2+)-ATPase or on surrounding lipids, decreases tryptophan fluorescence level of the protein. As much as 25% of the tryptophan fluorescence of the Ca(2+)-ATPase is quenched by Ruthenium red (with a dissociation constant of 100 nM), tryptophan residues located near the bilayer being preferentially affected.

MeSH terms

  • Animals
  • Biological Transport
  • Calcimycin / pharmacology
  • Calcium / metabolism
  • Calcium-Transporting ATPases / chemistry*
  • Cations, Divalent
  • Fluorescence
  • Magnesium / metabolism
  • Rabbits
  • Ruthenium Red / pharmacology*
  • Sarcoplasmic Reticulum / drug effects*
  • Sarcoplasmic Reticulum / enzymology

Substances

  • Cations, Divalent
  • Ruthenium Red
  • Calcimycin
  • Calcium-Transporting ATPases
  • Magnesium
  • Calcium