Abstract
We have previously shown that abstinence from morphine by either abrupt (AW) or precipitated (PW) withdrawal induces greater than 80% suppression in the capacity to mount an in vitro plaque-forming cell (PFC) response to sheep red blood cells at 24-h post withdrawal. Present studies on the mechanisms of immunosuppression showed that addition of normal unfractionated spleen cells, macrophage-enriched adherent cells, or CD11b(+) purified macrophages, to spleen cells taken from withdrawn mice, restored immune responses. Spleen cells from mice undergoing withdrawal also had decreased splenic mRNA and/or protein levels of IL-1beta, IL-1Ra, TNF-alpha, IL-12, and IFN-gamma. Addition of IL-1beta or IFN-gamma to AW cultures was able to reverse their immunosuppression. These results strongly suggest that morphine withdrawal results in a deficit of macrophage function.
Publication types
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Research Support, U.S. Gov't, P.H.S.
MeSH terms
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Animals
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Antigen Presentation / drug effects
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Antigen Presentation / immunology
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Antigens, CD / biosynthesis
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Apoptosis / drug effects
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Apoptosis / immunology
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B7-1 Antigen* / metabolism
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B7-2 Antigen
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Cells, Cultured
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Coculture Techniques
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Cytokines / antagonists & inhibitors*
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Cytokines / biosynthesis
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Cytokines / genetics
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Female
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Hemolytic Plaque Technique
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Immunosuppressive Agents / adverse effects*
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Leukocyte Count
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Macrophage Activation / drug effects
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Macrophage Activation / immunology
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Macrophages / drug effects
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Macrophages / immunology*
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Macrophages / metabolism
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Membrane Glycoproteins / antagonists & inhibitors*
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Membrane Glycoproteins / biosynthesis
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Mice
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Mice, Inbred C3H
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Morphine / adverse effects*
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RNA, Messenger / antagonists & inhibitors
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RNA, Messenger / biosynthesis
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Spleen / cytology
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Spleen / drug effects
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Spleen / immunology*
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Spleen / metabolism
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Substance Withdrawal Syndrome / immunology*
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Substance Withdrawal Syndrome / metabolism
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Time Factors
Substances
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Antigens, CD
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B7-1 Antigen
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B7-2 Antigen
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Cd86 protein, mouse
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Cytokines
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Immunosuppressive Agents
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Membrane Glycoproteins
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RNA, Messenger
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Morphine