D-fenfluramine-induced prolactin and cortisol release in major depression: response to treatment

J Affect Disord. 1992 Nov;26(3):143-50. doi: 10.1016/0165-0327(92)90009-u.

Abstract

In order to test the effects of various biological treatments on serotonergic function in depression, twenty-one patients with a diagnosis of major depression underwent neuroendocrine challenge tests before and after treatment with either ECT, fluoxetine or amitriptyline. The serotonin (5-HT) releasing agent d-fenfluramine was used as a challenge drug and cortisol (CORT) and prolactin (PRL) plasma levels were monitored over a 5-h period. Overall PRL responses were significantly enhanced following pharmacotherapy irrespective of therapeutic outcome. Effective treatment in each case lowered baseline CORT levels but CORT response to d-fenfluramine remained blunted. Hypercortisolaemia may be involved in the impaired pretreatment PRL response as a strong inverse relationship was established, for the combined studies, between basal CORT plasma concentrations and PRL responses.

Publication types

  • Clinical Trial
  • Comparative Study
  • Randomized Controlled Trial

MeSH terms

  • Adolescent
  • Adult
  • Amitriptyline / therapeutic use
  • Depressive Disorder / physiopathology
  • Depressive Disorder / psychology
  • Depressive Disorder / therapy*
  • Electroconvulsive Therapy
  • Female
  • Fenfluramine*
  • Fluoxetine / therapeutic use
  • Humans
  • Hydrocortisone / blood*
  • Male
  • Middle Aged
  • Personality Inventory
  • Prognosis
  • Prolactin / blood*
  • Receptors, Serotonin / drug effects
  • Receptors, Serotonin / physiology
  • Serotonin / physiology

Substances

  • Receptors, Serotonin
  • Fluoxetine
  • Amitriptyline
  • Fenfluramine
  • Serotonin
  • Prolactin
  • Hydrocortisone