Abstract
The ubiquitous ATP binding site offers a global target for protein kinase inhibitors. The corollary is that molecular selectivity with such agents may be difficult to achieve and ascertain. A relevant example is discussed in terms of design and biomedical rationale.
MeSH terms
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Adenosine Triphosphate / metabolism
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Binding Sites
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Cyclin-Dependent Kinases / antagonists & inhibitors*
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Cyclin-Dependent Kinases / chemistry*
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Cyclin-Dependent Kinases / genetics
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Cyclin-Dependent Kinases / metabolism
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Enzyme Inhibitors / chemistry*
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Enzyme Inhibitors / pharmacology*
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Glycogen Synthase Kinase 3 / antagonists & inhibitors*
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Glycogen Synthase Kinase 3 / chemistry*
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Glycogen Synthase Kinase 3 / genetics
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Glycogen Synthase Kinase 3 / metabolism
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Models, Molecular
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Protein Structure, Tertiary
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Substrate Specificity
Substances
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Enzyme Inhibitors
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Adenosine Triphosphate
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Cyclin-Dependent Kinases
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Glycogen Synthase Kinase 3